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Kidney International|September 18, 1997
Hypoxia stimulates proximal tubular cell matrix production via a TGF-beta1-independent mechanismC Orphanides, L G Fine, J T NormanKidney International. Supplement|June 1, 2000
Is there a common mechanism for the progression of different types of renal diseases other than proteinuria? Towards the unifying theme of chronic hypoxiaL G Fine, D Bandyopadhay, J T NormanKidney International|January 1, 1996
Pexicrine effects of basement membrane components on paracrine signaling by renal tubular cellsM P Lewis, L G Fine, J T NormanJournal of the American Society of Nephrology : JASN|January 1, 1992
Evolving role of growth factors in the renal response to acute and chronic diseaseL G Fine, M R Hammerman, H E AbboudKidney International. Supplement|January 1, 1993
Gene transfer into the mammalian kidney: first steps towards renal gene therapyA S Woolf, R J Bosch, L G FineEuropean Journal of Clinical Investigation|May 1, 1993
Mechanisms of tubulo-interstitial injury in progressive renal diseasesL G Fine, A C Ong, J T NormanExperimental Nephrology|January 1, 1993
Gene transfer into the mammalian kidney: direct retrovirus-transduction of regenerating tubular epithelial cellsR J Bosch, A S Woolf, L G FineJournal of Hypertension. Supplement : Official Journal of the International Society of Hypertension|April 1, 1992
Growth factors in the pathogenesis of renovascular complications of diabetes mellitusA S Woolf, R J Bosch, L G FineExperimental Nephrology|January 1, 1993
Gene transfer into the mammalian kidney: microtransplantation of retrovirus-transduced metanephric tissueA S Woolf, R J Bosch, L G FineThe American Journal of Physiology|January 1, 1978
Vasopressin responsiveness of renal adenylate cyclase in newborn rats and rabbitsD Schlondorff, H Weber, W Trizna, et al.Pageof 65