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The Journal of Membrane Biology|March 1, 1992
Differential response of DPI-modified cardiac Na+ channels to antiarrhythmic drugs: no flicker blockade by lidocaineI Benz, M KohlhardtThe Journal of Membrane Biology|May 1, 1994
Chemically modified cardiac Na+ channels and their sensitivity to antiarrhythmics: is there a hidden drug receptor?I Benz, M KohlhardtNaunyn-Schmiedeberg'S Archives of Pharmacology|September 24, 1975
The influence of metabolic inhibitors upon the transmembrane slow inward current in the mammalian ventricular myocardiumM Kohlhardt, M KüblerEuropean Journal of Pharmacology|October 26, 1988
Inhibitory effects of diprafenone stereoenantiomers on cardiac Na+ channelsM Kohlhardt, H FichtnerExperientia|February 15, 1976
The response of Ca-mediated action potentials and contractile activity in mammalian ventricular myocardium towards alkalosisM Kohlhardt, K HaapEuropean Biophysics Journal : EBJ|January 1, 1994
Blockade of cardiac outwardly rectifying K+ channels by TEA and class III antiarrhythmics--evidence against a single drug-sensitive channel siteI Benz, M KohlhardtEuropean Biophysics Journal : EBJ|January 1, 1992
Differential sensitivity of cardiac K+(ATP) channels to guanine nucleotides--evidence for a heterogeneous channel populationI Benz, M KohlhardtNaunyn-Schmiedeberg'S Archives of Pharmacology|July 18, 1977
Inhibition of the slow inward current by nifedipine in mammalian ventricular myocardiumM Kohlhardt, A FleckensteinThe Journal of Membrane Biology|May 1, 1988
Block of single cardiac Na+ channels by antiarrhythmic drugs: the effect of amiodarone, propafenone and diprafenoneM Kohlhardt, H FichtnerThe Journal of Membrane Biology|June 1, 1991
Responsiveness of cardiac Na+ channels to antiarrhythmic drugs: the role of inactivationI Benz, M KohlhardtPageof 5