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The Journal of Membrane Biology|November 1, 1992
Opposite effects of angiotensin II and the protein kinase C activator OAG on cardiac Na+ channelsI Benz, J W Herzig, M Kohlhardt
The American Journal of Physiology|October 1, 1988
Hyperpolarization and increased free calcium in acetylcholine-stimulated endothelial cellsR Busse, H Fichtner, A Lückhoff, et al.
Pflugers Archiv : European Journal of Physiology|March 22, 1975
Kinetics of inactivation and recovery of the slow inward current in the mammalian ventricular myocardiumM Kohlhardt, H Krause, M Kübler, et al.
The Journal of Membrane Biology|January 1, 1987
Single nonselective cation channels and Ca2+-activated K+ channels in aortic endothelial cellsH Fichtner, U Fröbe, R Busse, et al.
Circulation Research|May 1, 1989
On the mechanism of drug-induced blockade of Na+ currents: interaction of antiarrhythmic compounds with DPI-modified single cardiac Na+ channelsM Kohlhardt, H Fichtner, U Fröbe, et al.
The Journal of Membrane Biology|June 1, 1993
Responsiveness of cardiac Na+ channels to a site-directed antiserum against the cytosolic linker between domains III and IV and their sensitivity to other modifying agentsW Beck, I Benz, W Bessler, et al.
Biochimica Et Biophysica Acta|June 12, 1994
Conformational mapping of the cytosolic linker between domains III and IV of the cardiac Na+ channel protein and binding studies with a site-directed channel modifying antibodyW Beck, G Jung, W G Bessler, et al.
European Biophysics Journal : EBJ|January 1, 1997
Two types of modified cardiac Na+ channels after cytosolic interventions at the alpha-subunit capable of removing Na+ inactivationI Benz, W Beck, W Kraas, et al.
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