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Annals of the New York Academy of Sciences
|
June 8, 2000
Transgenic mice lacking white fat: models for understanding human lipoatrophic diabetes
M L Reitman, M M Mason, J Moitra, et al.
The Journal of Clinical Investigation
|
November 22, 2000
Adipose tissue is required for the antidiabetic, but not for the hypolipidemic, effect of thiazolidinediones
L Chao, B Marcus-Samuels, M M Mason, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
December 10, 1999
Torpor in mice is induced by both leptin-dependent and -independent mechanisms
O Gavrilova, L R Leon, B Marcus-Samuels, et al.
The Journal of Clinical Investigation
|
February 17, 2000
Surgical implantation of adipose tissue reverses diabetes in lipoatrophic mice
O Gavrilova, B Marcus-Samuels, D Graham, et al.
The Journal of Clinical Investigation
|
March 11, 2000
Paternal versus maternal transmission of a stimulatory G-protein alpha subunit knockout produces opposite effects on energy metabolism
S Yu, O Gavrilova, H Chen, et al.
Diabetologia
|
March 29, 2011
Increasing skeletal muscle fatty acid transport protein 1 (FATP1) targets fatty acids to oxidation and does not predispose mice to diet-induced insulin resistance
G P Holloway, C J Chou, J Lally, et al.
Molecular and Cellular Biology
|
June 24, 2000
Growth, adipose, brain, and skin alterations resulting from targeted disruption of the mouse peroxisome proliferator-activated receptor beta(delta)
J M Peters, S S Lee, W Li, et al.
Genes & Development
|
October 24, 1998
Life without white fat: a transgenic mouse
J Moitra, M M Mason, M Olive, et al.
Diabetes
|
May 26, 2001
Transgenic overexpression of leptin rescues insulin resistance and diabetes in a mouse model of lipoatrophic diabetes
K Ebihara, Y Ogawa, H Masuzaki, et al.
American Journal of Human Genetics
|
July 1, 1982
Two clonal cell populations (mosaicism) in a 46,XY male with mucolipidosis II (I-cell disease)--an autosomal recessive disorder
G H Thomas, C S Miller, K E Toomey, et al.
Page
of 4
Search research articles
Search
Showing results (21-30 of 35) with videos related to
Sort By:
Page
of 4
Annals of the New York Academy of Sciences
|
June 8, 2000
Transgenic mice lacking white fat: models for understanding human lipoatrophic diabetes
M L Reitman, M M Mason, J Moitra, et al.
The Journal of Clinical Investigation
|
November 22, 2000
Adipose tissue is required for the antidiabetic, but not for the hypolipidemic, effect of thiazolidinediones
L Chao, B Marcus-Samuels, M M Mason, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
December 10, 1999
Torpor in mice is induced by both leptin-dependent and -independent mechanisms
O Gavrilova, L R Leon, B Marcus-Samuels, et al.
The Journal of Clinical Investigation
|
February 17, 2000
Surgical implantation of adipose tissue reverses diabetes in lipoatrophic mice
O Gavrilova, B Marcus-Samuels, D Graham, et al.
The Journal of Clinical Investigation
|
March 11, 2000
Paternal versus maternal transmission of a stimulatory G-protein alpha subunit knockout produces opposite effects on energy metabolism
S Yu, O Gavrilova, H Chen, et al.
Diabetologia
|
March 29, 2011
Increasing skeletal muscle fatty acid transport protein 1 (FATP1) targets fatty acids to oxidation and does not predispose mice to diet-induced insulin resistance
G P Holloway, C J Chou, J Lally, et al.
Molecular and Cellular Biology
|
June 24, 2000
Growth, adipose, brain, and skin alterations resulting from targeted disruption of the mouse peroxisome proliferator-activated receptor beta(delta)
J M Peters, S S Lee, W Li, et al.
Genes & Development
|
October 24, 1998
Life without white fat: a transgenic mouse
J Moitra, M M Mason, M Olive, et al.
Diabetes
|
May 26, 2001
Transgenic overexpression of leptin rescues insulin resistance and diabetes in a mouse model of lipoatrophic diabetes
K Ebihara, Y Ogawa, H Masuzaki, et al.
American Journal of Human Genetics
|
July 1, 1982
Two clonal cell populations (mosaicism) in a 46,XY male with mucolipidosis II (I-cell disease)--an autosomal recessive disorder
G H Thomas, C S Miller, K E Toomey, et al.
Page
of 4