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M L Standaert

Showing results (61-70 of 80) with videos related to

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Metabolism: Clinical and Experimental|November 1, 1990
Protein kinase C inhibitors block insulin and PMA-stimulated hexose transport in isolated rat adipocytes and BC3H-1 myocytesM L Standaert, D J Buckley, T Ishizuka, et al.
The Journal of Biological Chemistry|January 24, 1997
Activation of protein kinase C (alpha, beta, and zeta) by insulin in 3T3/L1 cells. Transfection studies suggest a role for PKC-zeta in glucose transportG Bandyopadhyay, M L Standaert, L Zhao, et al.
The Biochemical Journal|November 15, 1988
Mechanisms whereby insulin increases diacylglycerol in BC3H-1 myocytesR V Farese, D R Cooper, T S Konda, et al.
The Journal of Clinical Investigation|May 1, 1994
Preferential activation of microsomal diacylglycerol/protein kinase C signaling during glucose treatment (De Novo phospholipid synthesis) of rat adipocytesR V Farese, M L Standaert, T P Arnold, et al.
Diabetes|October 1, 1996
Chronic activation of protein kinase C in soleus muscles and other tissues of insulin-resistant type II diabetic Goto-Kakizaki (GK), obese/aged, and obese/Zucker rats. A mechanism for inhibiting glycogen synthesisA Avignon, K Yamada, X Zhou, et al.
The Journal of Clinical Endocrinology and Metabolism|August 5, 2004
Impaired activation of protein kinase C-zeta by insulin and phosphatidylinositol-3,4,5-(PO4)3 in cultured preadipocyte-derived adipocytes and myotubes of obese subjectsM P Sajan, M L Standaert, A Miura, et al.
The Journal of Biological Chemistry|July 21, 2001
Glucose activates protein kinase C-zeta /lambda through proline-rich tyrosine kinase-2, extracellular signal-regulated kinase, and phospholipase D: a novel mechanism for activating glucose transporter translocationG Bandyopadhyay, M P Sajan, Y Kanoh, et al.
FEBS Letters|April 6, 1987
Insulin but not phorbol ester treatment increases phosphorylation of vinculin by protein kinase C in BC3H-1 myocytesD R Cooper, C M de Ruiz Galaretta, L F Fanjul, et al.
Diabetologia|October 31, 2008
Combined thiazolidinedione-metformin treatment synergistically improves insulin signalling to insulin receptor substrate-1-dependent phosphatidylinositol 3-kinase, atypical protein kinase C and protein kinase B/Akt in human diabetic muscleN Temofonte, M P Sajan, S Nimal, et al.
The Biochemical Journal|October 15, 1985
The de novo phospholipid effect of insulin is associated with increases in diacylglycerol, but not inositol phosphates or cytosolic Ca2+R V Farese, J S Davis, D E Barnes, et al.
Pageof 8

Showing results (61-70 of 80) with videos related to

Sort By:
Pageof 8
Metabolism: Clinical and Experimental|November 1, 1990
Protein kinase C inhibitors block insulin and PMA-stimulated hexose transport in isolated rat adipocytes and BC3H-1 myocytesM L Standaert, D J Buckley, T Ishizuka, et al.
The Journal of Biological Chemistry|January 24, 1997
Activation of protein kinase C (alpha, beta, and zeta) by insulin in 3T3/L1 cells. Transfection studies suggest a role for PKC-zeta in glucose transportG Bandyopadhyay, M L Standaert, L Zhao, et al.
The Biochemical Journal|November 15, 1988
Mechanisms whereby insulin increases diacylglycerol in BC3H-1 myocytesR V Farese, D R Cooper, T S Konda, et al.
The Journal of Clinical Investigation|May 1, 1994
Preferential activation of microsomal diacylglycerol/protein kinase C signaling during glucose treatment (De Novo phospholipid synthesis) of rat adipocytesR V Farese, M L Standaert, T P Arnold, et al.
Diabetes|October 1, 1996
Chronic activation of protein kinase C in soleus muscles and other tissues of insulin-resistant type II diabetic Goto-Kakizaki (GK), obese/aged, and obese/Zucker rats. A mechanism for inhibiting glycogen synthesisA Avignon, K Yamada, X Zhou, et al.
The Journal of Clinical Endocrinology and Metabolism|August 5, 2004
Impaired activation of protein kinase C-zeta by insulin and phosphatidylinositol-3,4,5-(PO4)3 in cultured preadipocyte-derived adipocytes and myotubes of obese subjectsM P Sajan, M L Standaert, A Miura, et al.
The Journal of Biological Chemistry|July 21, 2001
Glucose activates protein kinase C-zeta /lambda through proline-rich tyrosine kinase-2, extracellular signal-regulated kinase, and phospholipase D: a novel mechanism for activating glucose transporter translocationG Bandyopadhyay, M P Sajan, Y Kanoh, et al.
FEBS Letters|April 6, 1987
Insulin but not phorbol ester treatment increases phosphorylation of vinculin by protein kinase C in BC3H-1 myocytesD R Cooper, C M de Ruiz Galaretta, L F Fanjul, et al.
Diabetologia|October 31, 2008
Combined thiazolidinedione-metformin treatment synergistically improves insulin signalling to insulin receptor substrate-1-dependent phosphatidylinositol 3-kinase, atypical protein kinase C and protein kinase B/Akt in human diabetic muscleN Temofonte, M P Sajan, S Nimal, et al.
The Biochemical Journal|October 15, 1985
The de novo phospholipid effect of insulin is associated with increases in diacylglycerol, but not inositol phosphates or cytosolic Ca2+R V Farese, J S Davis, D E Barnes, et al.
Pageof 8