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Journal of Immunology (Baltimore, Md. : 1950)|April 20, 2006
A role for the MutL mismatch repair Mlh3 protein in immunoglobulin class switch DNA recombination and somatic hypermutationXiaoping Wu, Connie Y Tsai, Marienida B Patam, et al.
Doklady Biological Sciences : Proceedings of the Academy of Sciences of the USSR, Biological Sciences Sections|September 7, 2016
Specific effect of the HLDF differentiation factor on the cytokine production potential of immunocompetent blood cells in stomach adenocarcinomaA I Autenshlyus, T A Kunts, E S Mikhaylova, et al.
Biomedical Science|March 1, 1990
Site-directed mutagenesis of the inhibitory subunit of retinal rod cyclic GMP phosphodiesteraseV M Lipkin, I P Udovichenko, V A Bondarenko, et al.
FEBS Letters|February 26, 1996
Human cone-specific cGMP phosphodiesterase alpha' subunit: complete cDNA sequence and gene arrangementE A Feshchenko, S G Andreeva, V A Suslova, et al.
Journal of the National Cancer Institute|May 1, 1986
Expression of murine gamma fetal antigen in adult hematopoietic tissue and during induced differentiation of Friend erythroleukemia cellsP J Higgins, A E Silverstone, C Bueti, et al.
Biochemistry. Biokhimiia|March 10, 2024
Topology of Ubiquitin Chains in the Chromatosomal Environment of the E3 Ubiquitin Ligase RNF168Anna A Kudriaeva, Lyudmila A Yakubova, George A Saratov, et al.
Cancer Letters|June 19, 2001
Inhibition by dietary dibenzoylmethane of mammary gland proliferation, formation of DMBA-DNA adducts in mammary glands, and mammary tumorigenesis in Sencar miceC C Lin, Y P Lu, Y R Lou, et al.
Journal of the National Cancer Institute|May 1, 1983
Reduced capacity for DNA repair synthesis in patients with or genetically predisposed to colorectal cancerR W Pero, D G Miller, M Lipkin, et al.
Bioorganicheskaia Khimiia|July 1, 1996
[Preparation of the myristoylated and nonmyristoylated form of recombinant recoverin in E. coli cells and comparison of their functional activity]A A Zargarov, I I Senin, A M Alekseev, et al.
Cancer Research|February 11, 2010
NOTCH signaling is required for formation and self-renewal of tumor-initiating cells and for repression of secretory cell differentiation in colon cancerShaheen S Sikandar, Kira T Pate, Scott Anderson, et al.
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