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Aging Cell|February 9, 2017
The oxidized thiol proteome in aging and cataractous mouse and human lens revealed by ICAT labelingBenlian Wang, Grant Hom, Sheng Zhou, et al.
Bioresearch Open Access|August 27, 2015
Biological Effects Induced by Specific Advanced Glycation End Products in the Reconstructed Skin Model of AgingHervé Pageon, Hélène Zucchi, Zhenyu Dai, et al.
The Journal of Biological Chemistry|January 29, 2005
Glucosepane is a major protein cross-link of the senescent human extracellular matrix. Relationship with diabetesDavid R Sell, Klaus M Biemel, Oliver Reihl, et al.
Kidney International|February 1, 1996
Monocyte/macrophage response to beta 2-microglobulin modified with advanced glycation end productsT Miyata, Y Iida, Y Ueda, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 19, 1996
Identification of pentosidine as a native structure for advanced glycation end products in beta-2-microglobulin-containing amyloid fibrils in patients with dialysis-related amyloidosisT Miyata, S Taneda, R Kawai, et al.
Neuroscience Letters|August 8, 1997
Advanced glycation modification of Rosenthal fibers in patients with Alexander diseaseR J Castellani, G Perry, P L Harris, et al.
Molecular and Cellular Biochemistry|August 26, 2006
Relative suppression of the sodium-dependent Vitamin C transport in mouse versus human lens epithelial cellsMark E Obrenovich, Xingjun Fan, Makoto Satake, et al.
Diabetes/Metabolism Reviews|December 1, 1991
Pentosidine: a molecular marker for the cumulative damage to proteins in diabetes, aging, and uremiaD R Sell, R H Nagaraj, S K Grandhee, et al.
Diabetes|October 27, 2005
Glycation and carboxymethyllysine levels in skin collagen predict the risk of future 10-year progression of diabetic retinopathy and nephropathy in the diabetes control and complications trial and epidemiology of diabetes interventions and complications participants with type 1 diabetesSaul Genuth, Wanjie Sun, Patricia Cleary, et al.
Human Molecular Genetics|February 4, 2017
Gclc deficiency in mouse CNS causes mitochondrial damage and neurodegenerationWeiyi Feng, Mariana Rosca, Yuxuan Fan, et al.
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