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Frontiers in Veterinary Science|July 29, 2025
Reduced glomerular and elevated tubulointerstitial transglutaminase pathway and its inhibition in a rat model of renal warm ischemia: implications for feline chronic kidney diseaseA C Sánchez-Lara, M Maamra, J L Haylor
The Journal of Clinical Endocrinology and Metabolism|September 10, 2004
Pegvisomant, a growth hormone-specific antagonist, undergoes cellular internalizationM Maamra, J J Kopchick, C J Strasburger, et al.
The Journal of Biological Chemistry|May 18, 1999
Studies with a growth hormone antagonist and dual-fluorescent confocal microscopy demonstrate that the full-length human growth hormone receptor, but not the truncated isoform, is very rapidly internalized independent of Jak2-Stat5 signalingM Maamra, J Finidori, S Von Laue, et al.
Endocrinology|September 21, 2001
Generation of human soluble leptin receptor by proteolytic cleavage of membrane-anchored receptorsM Maamra, M Bidlingmaier, M C Postel-Vinay, et al.
The Journal of Clinical Endocrinology and Metabolism|April 12, 2001
Binding and functional studies with the growth hormone receptor antagonist, B2036-PEG (pegvisomant), reveal effects of pegylation and evidence that it binds to a receptor dimerR J Ross, K C Leung, M Maamra, et al.
The Journal of Endocrinology|May 16, 2000
Stimulation of endogenous GH and interleukin-6 receptors selectively activates different Jaks and Stats, with a Stat5 specific synergistic effect of dexamethasoneS von Laue, J Finidori, M Maamra, et al.
The Journal of Endocrinology|February 8, 2006
A 36 residues insertion in the dimerization domain of the growth hormone receptor results in defective trafficking rather than impaired signalingM Maamra, A Milward, H Zarkesh Esfahani, et al.
The Journal of Endocrinology|April 20, 2001
Differential expression of suppressors of cytokine signalling genes in response to nutrition and growth hormone in the septic ratT S Johnson, M O'Leary, S K Justice, et al.
The Journal of Clinical Endocrinology and Metabolism|March 6, 2004
Growth hormone (GH) insensitivity syndrome due to a GH receptor truncated after Box1, resulting in isolated failure of STAT 5 signal transductionA Milward, L Metherell, M Maamra, et al.
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