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Developmental Cell|December 17, 2008
UPR pathways combine to prevent hepatic steatosis caused by ER stress-mediated suppression of transcriptional master regulatorsD Thomas Rutkowski, Jun Wu, Sung-Hoon Back, et al.
Nature|July 8, 2016
TTC39B deficiency stabilizes LXR reducing both atherosclerosis and steatohepatitisJoanne Hsieh, Masahiro Koseki, Matthew M Molusky, et al.
Circulation|May 22, 2015
Pathogenesis of the Novel Autoimmune-Associated Long-QT SyndromeYuankun Yue, Monica Castrichini, Ujala Srivastava, et al.
The Journal of Clinical Investigation|April 23, 2026
The N-terminus of Apolipoprotein B mediates the interaction of atherogenic lipoproteins with endothelial cellsAinara G Cabodevilla, Camila Calistru, Waqas Younis, et al.
Science (New York, N.Y.)|August 31, 2023
Intracellular tPA-PAI-1 interaction determines VLDL assembly in hepatocytesWen Dai, Heng Zhang, Hayley Lund, et al.
Journal of Clinical Lipidology|October 15, 2022
Guidance for the diagnosis and treatment of hypolipidemia disordersCindy Bredefeld, M Mahmood Hussain, Maurizio Averna, et al.
Hepatology (Baltimore, Md.)|September 2, 2022
Loss of hepatic SMLR1 causes hepatosteatosis and protects against atherosclerosis due to decreased hepatic VLDL secretionWillemien van Zwol, Antoine Rimbert, Justina C Wolters, et al.
Physical Review Letters|April 16, 2016
Anisotropic Flow of Charged Particles in Pb-Pb Collisions at √[s{NN}]=5.02 TeVJ Adam, D Adamová, M M Aggarwal, et al.
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