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AMIA ... Annual Symposium Proceedings. AMIA Symposium|January 20, 2004
A patient-record supporting treatment cost determinationB Spyropoulos, M MarinisThe Journal of Biological Chemistry|November 25, 2010
A novel motif in the Crohn's disease susceptibility protein, NOD2, allows TRAF4 to down-regulate innate immune responsesJill M Marinis, Craig R Homer, Christine McDonald, et al.Current Biology : CB|September 9, 2008
MEKK4 sequesters RIP2 to dictate NOD2 signal specificityNivedita M Clark, Jill M Marinis, Brian A Cobb, et al.Current Biology : CB|July 14, 2009
ITCH K63-ubiquitinates the NOD2 binding protein, RIP2, to influence inflammatory signaling pathwaysMingfang Tao, Peter C Scacheri, Jill M Marinis, et al.Molecular and Cellular Biology|May 2, 2012
IκB kinase α phosphorylation of TRAF4 downregulates innate immune signalingJill M Marinis, Jessica E Hutti, Craig R Homer, et al.Molecular Cell|September 10, 2014
Integrator regulates transcriptional initiation and pause release following activationAlessandro Gardini, David Baillat, Matteo Cesaroni, et al.Cell Reports|July 23, 2013
Innate immune-directed NF-κB signaling requires site-specific NEMO ubiquitinationJanice C Jun, Sylvia Kertesy, Mark B Jones, et al.Genes & Development|May 3, 2014
Anti-diabetic rosiglitazone remodels the adipocyte transcriptome by redistributing transcription to PPARγ-driven enhancersSonia E Step, Hee-Woong Lim, Jill M Marinis, et al.Genes & Development|July 15, 2018
PPARγ is a nexus controlling alternative activation of macrophages via glutamine metabolismVictoria L Nelson, Hoang C B Nguyen, Juan C Garcìa-Cañaveras, et al.The Journal of Experimental Medicine|November 2, 2017
Autophagy protein ATG16L1 prevents necroptosis in the intestinal epitheliumYu Matsuzawa-Ishimoto, Yusuke Shono, Luis E Gomez, et al.Pageof 2