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Cancer Discovery|January 10, 2020
Selective Inhibition of HDAC3 Targets Synthetic Vulnerabilities and Activates Immune Surveillance in LymphomaPatrizia Mondello, Saber Tadros, Matt Teater, et al.Nature|December 10, 2020
Histone H1 loss drives lymphoma by disrupting 3D chromatin architectureNevin Yusufova, Andreas Kloetgen, Matt Teater, et al.Iscience|December 30, 2020
Cell-free DNA (cfDNA) and Exosome Profiling from a Year-Long Human Spaceflight Reveals Circulating BiomarkersDaniela Bezdan, Kirill Grigorev, Cem Meydan, et al.Cancer Discovery|October 14, 2016
CREBBP Inactivation Promotes the Development of HDAC3-Dependent LymphomasYanwen Jiang, Ana Ortega-Molina, Huimin Geng, et al.Proceedings of the National Academy of Sciences of the United States of America|May 16, 2025
Mutant IDH1 cooperates with NPM1c or FLT3ITD to drive distinct myeloid diseases and molecular outcomesTakashi Sakamoto, Julie Leca, Xin Zhang, et al.Cancer Discovery|October 3, 2018
TET2 Deficiency Causes Germinal Center Hyperplasia, Impairs Plasma Cell Differentiation, and Promotes B-cell LymphomagenesisPilar M Dominguez, Hussein Ghamlouch, Wojciech Rosikiewicz, et al.Nature Medicine|June 21, 2016
Distinct evolution and dynamics of epigenetic and genetic heterogeneity in acute myeloid leukemiaSheng Li, Francine E Garrett-Bakelman, Stephen S Chung, et al.Blood Neoplasia|January 29, 2026
The LMO2-LDB1-TAL1 complex regulates transcription networks in acute myeloid leukemiaNicholas Dunham, Zhenjia Wang, Yaseswini Neelamraju, et al.Blood Cancer Discovery|November 8, 2022
Distinct Genetically Determined Origins of Myd88/BCL2-Driven Aggressive Lymphoma Rationalize Targeted Therapeutic Intervention StrategiesRuth Flümann, Julia Hansen, Benedikt W Pelzer, et al.Blood Cancer Discovery|January 15, 2021
An Autochthonous Mouse Model of Myd88- and BCL2-Driven Diffuse Large B-cell Lymphoma Reveals Actionable Molecular VulnerabilitiesRuth Flümann, Tim Rehkämper, Pascal Nieper, et al.Pageof 37