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Nature Medicine
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June 21, 2016
Distinct evolution and dynamics of epigenetic and genetic heterogeneity in acute myeloid leukemia
Sheng Li, Francine E Garrett-Bakelman, Stephen S Chung, et al.
Blood Neoplasia
|
January 29, 2026
The LMO2-LDB1-TAL1 complex regulates transcription networks in acute myeloid leukemia
Nicholas Dunham, Zhenjia Wang, Yaseswini Neelamraju, et al.
Blood Cancer Discovery
|
November 8, 2022
Distinct Genetically Determined Origins of Myd88/BCL2-Driven Aggressive Lymphoma Rationalize Targeted Therapeutic Intervention Strategies
Ruth Flümann, Julia Hansen, Benedikt W Pelzer, et al.
Blood Cancer Discovery
|
January 15, 2021
An Autochthonous Mouse Model of Myd88- and BCL2-Driven Diffuse Large B-cell Lymphoma Reveals Actionable Molecular Vulnerabilities
Ruth Flümann, Tim Rehkämper, Pascal Nieper, et al.
Cancer Cell
|
May 18, 2013
EZH2 is required for germinal center formation and somatic EZH2 mutations promote lymphoid transformation
Wendy Béguelin, Relja Popovic, Matt Teater, et al.
Science Translational Medicine
|
March 9, 2022
Blocking UBE2N abrogates oncogenic immune signaling in acute myeloid leukemia
Laura Barreyro, Avery M Sampson, Chiharu Ishikawa, et al.
The Journal of Clinical Investigation
|
April 5, 2022
Tumor-associated antigen PRAME exhibits dualistic functions that are targetable in diffuse large B cell lymphoma
Katsuyoshi Takata, Lauren C Chong, Daisuke Ennishi, et al.
Biorxiv : the Preprint Server for Biology
|
December 22, 2025
Signaling induced biophysical disruption of repressed chromatin domains drives immune cell fate
Alexia Martínez de Paz, Christopher R Chin, Mythili Ketavarapu, et al.
Cancer Cell
|
June 4, 2026
Pathogenesis of diffuse large B cell lymphoma proteogenotypes
Julius C Enssle, Björn Häupl, Arber Qoku, et al.
Nature Medicine
|
November 15, 2016
DNMT3A mutations promote anthracycline resistance in acute myeloid leukemia via impaired nucleosome remodeling
Olga A Guryanova, Kaitlyn Shank, Barbara Spitzer, et al.
Page
of 33
Search research articles
Search
Showing results (301-310 of 322) with videos related to
Sort By:
Page
of 33
Nature Medicine
|
June 21, 2016
Distinct evolution and dynamics of epigenetic and genetic heterogeneity in acute myeloid leukemia
Sheng Li, Francine E Garrett-Bakelman, Stephen S Chung, et al.
Blood Neoplasia
|
January 29, 2026
The LMO2-LDB1-TAL1 complex regulates transcription networks in acute myeloid leukemia
Nicholas Dunham, Zhenjia Wang, Yaseswini Neelamraju, et al.
Blood Cancer Discovery
|
November 8, 2022
Distinct Genetically Determined Origins of Myd88/BCL2-Driven Aggressive Lymphoma Rationalize Targeted Therapeutic Intervention Strategies
Ruth Flümann, Julia Hansen, Benedikt W Pelzer, et al.
Blood Cancer Discovery
|
January 15, 2021
An Autochthonous Mouse Model of Myd88- and BCL2-Driven Diffuse Large B-cell Lymphoma Reveals Actionable Molecular Vulnerabilities
Ruth Flümann, Tim Rehkämper, Pascal Nieper, et al.
Cancer Cell
|
May 18, 2013
EZH2 is required for germinal center formation and somatic EZH2 mutations promote lymphoid transformation
Wendy Béguelin, Relja Popovic, Matt Teater, et al.
Science Translational Medicine
|
March 9, 2022
Blocking UBE2N abrogates oncogenic immune signaling in acute myeloid leukemia
Laura Barreyro, Avery M Sampson, Chiharu Ishikawa, et al.
The Journal of Clinical Investigation
|
April 5, 2022
Tumor-associated antigen PRAME exhibits dualistic functions that are targetable in diffuse large B cell lymphoma
Katsuyoshi Takata, Lauren C Chong, Daisuke Ennishi, et al.
Biorxiv : the Preprint Server for Biology
|
December 22, 2025
Signaling induced biophysical disruption of repressed chromatin domains drives immune cell fate
Alexia Martínez de Paz, Christopher R Chin, Mythili Ketavarapu, et al.
Cancer Cell
|
June 4, 2026
Pathogenesis of diffuse large B cell lymphoma proteogenotypes
Julius C Enssle, Björn Häupl, Arber Qoku, et al.
Nature Medicine
|
November 15, 2016
DNMT3A mutations promote anthracycline resistance in acute myeloid leukemia via impaired nucleosome remodeling
Olga A Guryanova, Kaitlyn Shank, Barbara Spitzer, et al.
Page
of 33