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Plos One|April 13, 2011
Chronic intranasal treatment with an anti-Aβ(30-42) scFv antibody ameliorates amyloid pathology in a transgenic mouse model of Alzheimer's diseaseSusann Cattepoel, Michael Hanenberg, Luka Kulic, et al.FEBS Letters|December 13, 2000
Immature human NT2 cells grafted into mouse brain differentiate into neuronal and glial cell typesA Ferrari, E Ehler, R M Nitsch, et al.Journal of Plastic, Reconstructive & Aesthetic Surgery : JPRAS|April 22, 2008
In vivo perfusion analysis of normal and dysplastic ears and its implication on total auricular reconstructionH Frenzel, B Wollenberg, A Steffen, et al.Journal of Neural Transmission (Vienna, Austria : 1996)|March 3, 2004
Transgenic BACE expression in mouse neurons accelerates amyloid plaque pathologyM H Mohajeri, K D Saini, R M NitschAnnals of the New York Academy of Sciences|January 17, 1996
Regulation of APP processing. Potential for the therapeutical reduction of brain amyloid burdenR M Nitsch, R J Wurtman, J H GrowdonThe Journal of Biological Chemistry|July 10, 2002
Abeta 42-induced increase in neprilysin is associated with prevention of amyloid plaque formation in vivoM Hasan Mohajeri, M Axel Wollmer, Roger M NitschJournal of Neurochemistry|February 1, 1993
Characterization of membrane-bound and solubilized high-affinity binding sites for 5'-N-ethylcarboxamido[3H]adenosine from bovine cerebral cortexA Lorenzen, M Nitsch-Kirsch, H Vogt, et al.The American Journal of Pathology|August 26, 2003
Activation of the ERK and JNK signaling pathways caused by neuron-specific inhibition of PP2A in transgenic miceStefan Kins, Pascal Kurosinski, Roger M Nitsch, et al.Brain : a Journal of Neurology|December 1, 1992
The mysterious relocation of the bundle of TürckJ D Schmahmann, R M Nitsch, D N PandyaArzneimittel-Forschung|March 1, 1995
Regulation of proteolytic processing of the amyloid beta-protein precursor by first messengers. A novel potential approach for the treatment of Alzheimer's diseaseR M Nitsch, R J Wurtman, J H GrowdonPageof 27