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Neuroscience|June 23, 2004
The expression of PEA-15 (phosphoprotein enriched in astrocytes of 15 kDa) defines subpopulations of astrocytes and neurons throughout the adult mouse brainA Sharif, F Renault, F Beuvon, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 1, 1994
Transforming growth factor alpha (TGF alpha) expression in degenerating motoneurons of the murine mutant wobbler: a neuronal signal for astrogliosis?M P Junier, M Coulpier, N Le Forestier, et al.Proceedings of the National Academy of Sciences of the United States of America|November 1, 1991
Transforming growth factor alpha contributes to the mechanism by which hypothalamic injury induces precocious pubertyM P Junier, Y J Ma, M E Costa, et al.Endocrinology|March 1, 1990
Epoxygenase products of arachidonic acid are endogenous constituents of the hypothalamus involved in D2 receptor-mediated, dopamine-induced release of somatostatinM P Junier, F Dray, I Blair, et al.Molecular and Cellular Neurosciences|October 29, 1998
Regulation of growth factor gene expression in degenerating motoneurons of the murine mutant wobbler: a cellular patch-sampling/RT-PCR studyM P Junier, P Legendre, C V Esguerra, et al.Molecular and Cellular Neurosciences|June 1, 1994
Expression of epidermal growth factor receptor changes in the hypothalamus during the onset of female pubertyY J Ma, D F Hill, M P Junier, et al.Oncogene|October 24, 2006
Transforming growth factor alpha promotes sequential conversion of mature astrocytes into neural progenitors and stem cellsA Sharif, P Legendre, V Prévot, et al.Oncogene|March 15, 2006
Transforming growth factor alpha acts as a gliatrophin for mouse and human astrocytesA Sharif, V Prévot, F Renault-Mihara, et al.Molecular and Cellular Neurosciences|February 13, 2001
TTF-1, a homeodomain gene required for diencephalic morphogenesis, is postnatally expressed in the neuroendocrine brain in a developmentally regulated and cell-specific fashionB J Lee, G J Cho, R B Norgren, et al.Oncogene|September 20, 2017
DOCK4 promotes loss of proliferation in glioblastoma progenitor cells through nuclear beta-catenin accumulation and subsequent miR-302-367 cluster expressionD N Debruyne, L Turchi, F Burel-Vandenbos, et al.Pageof 3