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The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 1, 1993
Basic FGF regulates the expression of a functional 71 kDa NMDA receptor protein that mediates calcium influx and neurotoxicity in hippocampal neuronsM P Mattson, K N Kumar, H Wang, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 1, 1997
Amyloid beta-peptide impairs glucose transport in hippocampal and cortical neurons: involvement of membrane lipid peroxidationR J Mark, Z Pang, J W Geddes, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 1, 1992
beta-Amyloid peptides destabilize calcium homeostasis and render human cortical neurons vulnerable to excitotoxicityM P Mattson, B Cheng, D Davis, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 26, 1995
Tumor necrosis factors alpha and beta protect neurons against amyloid beta-peptide toxicity: evidence for involvement of a kappa B-binding factor and attenuation of peroxide and Ca2+ accumulationS W Barger, D Hörster, K Furukawa, et al.
Journal of Molecular Neuroscience : MN|June 15, 2000
The catalytic subunit of telomerase is expressed in developing brain neurons and serves a cell survival-promoting functionW Fu, M Killen, C Culmsee, et al.
The Journal of Biological Chemistry|April 15, 2000
Presenilin-1 mutations increase levels of ryanodine receptors and calcium release in PC12 cells and cortical neuronsS L Chan, M Mayne, C P Holden, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 23, 1997
The actin-severing protein gelsolin modulates calcium channel and NMDA receptor activities and vulnerability to excitotoxicity in hippocampal neuronsK Furukawa, W Fu, Y Li, et al.
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