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Neuroscience|November 28, 2002
Nerve growth factor survival signaling in cultured hippocampal neurons is mediated through TrkA and requires the common neurotrophin receptor P75C Culmsee, N Gerling, M Lehmann, et al.Neuroscience|August 12, 2008
Neuroprotection in stroke by complement inhibition and immunoglobulin therapyT V Arumugam, T M Woodruff, J D Lathia, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 1, 1997
Alzheimer's presenilin mutation sensitizes neural cells to apoptosis induced by trophic factor withdrawal and amyloid beta-peptide: involvement of calcium and oxyradicalsQ Guo, B L Sopher, K Furukawa, et al.The American Journal of Clinical Nutrition|March 1, 1996
Linoleic acid activates nuclear transcription factor-kappa B (NF-kappa B) and induces NF-kappa B-dependent transcription in cultured endothelial cellsB Hennig, M Toborek, S Joshi-Barve, et al.Journal of Neurochemistry|August 1, 1994
Secreted forms of beta-amyloid precursor protein protect against ischemic brain injuryV L Smith-Swintosky, L C Pettigrew, S D Craddock, et al.Journal of Neurochemistry|February 26, 1999
Increased vulnerability of hippocampal neurons from presenilin-1 mutant knock-in mice to amyloid beta-peptide toxicity: central roles of superoxide production and caspase activationQ Guo, L Sebastian, B L Sopher, et al.Neuromolecular Medicine|June 30, 2009
Amyloid-beta induces a caspase-mediated cleavage of P2X4 to promote purinotoxicityR Varma, Y Chai, J Troncoso, et al.Brain Research. Brain Research Reviews|February 1, 1997
Cellular signaling roles of TGF beta, TNF alpha and beta APP in brain injury responses and Alzheimer's diseaseM P Mattson, S W Barger, K Furukawa, et al.Nature Medicine|January 12, 1999
Increased vulnerability of hippocampal neurons to excitotoxic necrosis in presenilin-1 mutant knock-in miceQ Guo, W Fu, B L Sopher, et al.Science (New York, N.Y.)|December 22, 1999
Microglial activation resulting from CD40-CD40L interaction after beta-amyloid stimulationJ Tan, T Town, D Paris, et al.Pageof 34