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Diabetes|June 27, 2000
T-cell lines reactive to an immunodominant epitope of the tyrosine phosphatase-like autoantigen IA-2 in type 1 diabetesC J Hawkes, N C Schloot, J Marks, et al.Diabetes|July 1, 1994
Lack of immunohistological changes in the islets of nondiabetic, autoimmune, polyendocrine patients with beta-selective GAD-specific islet cell antibodiesR Wagner, J M McNally, E Bonifacio, et al.Diabetes|July 1, 1992
Antibodies to GAD and tryptic fragments of islet 64K antigen as distinct markers for development of IDDM. Studies with identical twinsM R Christie, R Y Tun, S S Lo, et al.Diabetologia|April 1, 1994
Slow metabolic deterioration towards diabetes in islet cell antibody positive patients with autoimmune polyendocrine diseaseR Wagner, S Genovese, E Bosi, et al.Diabetes Research (Edinburgh, Scotland)|February 1, 1989
Supravital dithizone staining in the isolation of human and rat pancreatic isletsW A Hansen, M R Christie, R Kahn, et al.Journal of Autoimmunity|May 19, 2009
Autoantibodies and associated T-cell responses to determinants within the 831-860 region of the autoantigen IA-2 in Type 1 diabetesS M Weenink, J Lo, C R Stephenson, et al.Diabetologia|July 28, 2007
GAD autoantibodies and epitope reactivities persist after diagnosis in latent autoimmune diabetes in adults but do not predict disease progression: UKPDS 77M Desai, C A Cull, V A Horton, et al.Pageof 5