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Atherosclerosis|December 4, 2003
Heterozygosity for ABCA1 gene mutations: effects on enzymes, apolipoproteins and lipoprotein particle sizeJ A Kuivenhoven, G K Hovingh, A van Tol, et al.Human Molecular Genetics|February 1, 1997
Contribution of DNA sequence and CAG size to mutation frequencies of intermediate alleles for Huntington disease: evidence from single sperm analysesS S Chong, E Almqvist, H Telenius, et al.The American Journal of Clinical Nutrition|March 1, 1987
Body composition of Peruvian children with short stature and high weight-for-height. I. Total body-water measurements and their prediction from anthropometric valuesT W Boutton, F L Trowbridge, M M Nelson, et al.Circulation|March 10, 2001
Common genetic variation in ABCA1 is associated with altered lipoprotein levels and a modified risk for coronary artery diseaseS M Clee, A H Zwinderman, J C Engert, et al.Pediatric Blood & Cancer|February 27, 2013
Validation of variants in SLC28A3 and UGT1A6 as genetic markers predictive of anthracycline-induced cardiotoxicity in childrenH Visscher, C J D Ross, S R Rassekh, et al.The Journal of Biological Chemistry|June 26, 2001
Human ABCA1 BAC transgenic mice show increased high density lipoprotein cholesterol and ApoAI-dependent efflux stimulated by an internal promoter containing liver X receptor response elements in intron 1R R Singaraja, V Bocher, E R James, et al.The Journal of Clinical Investigation|November 22, 2000
Age and residual cholesterol efflux affect HDL cholesterol levels and coronary artery disease in ABCA1 heterozygotesS M Clee, J J Kastelein, M van Dam, et al.Neuron|July 13, 1999
A YAC mouse model for Huntington's disease with full-length mutant huntingtin, cytoplasmic toxicity, and selective striatal neurodegenerationJ G Hodgson, N Agopyan, C A Gutekunst, et al.Arteriosclerosis, Thrombosis, and Vascular Biology|December 31, 1997
Ethnic variation and in vivo effects of the -93t-->g promoter variant in the lipoprotein lipase geneE Ehrenborg, S M Clee, S N Pimstone, et al.Cell Death and Differentiation|January 10, 2004
Specific caspase interactions and amplification are involved in selective neuronal vulnerability in Huntington's diseaseE Hermel, J Gafni, S S Propp, et al.Pageof 34