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Cardiovascular Research|December 12, 2001
NO-cGMP pathway increases the hyperpolarisation-activated current, I(f), and heart rate during adrenergic stimulationN Herring, L Rigg, D A Terrar, et al.British Journal of Pharmacology|September 1, 1993
The positive inotropic effect of compound II, a novel analogue of sotalol, on guinea-pig papillary muscles and single ventricular myocytesE White, S P Connors, E W Gill, et al.Journal of Medicinal Chemistry|May 1, 1991
The synthesis and potassium channel blocking activity of some (4-methanesulfonamidophenoxy)propanolamines as potential class III antiarrhythmic agentsS P Connors, P D Dennis, E W Gill, et al.Journal of Molecular and Cellular Cardiology|October 3, 2000
Role of cGMP-inhibited phosphodiesterase and sarcoplasmic calcium in mediating the increase in basal heart rate with nitric oxide donorsP Musialek, L Rigg, D A Terrar, et al.Current Biology : CB|August 1, 1996
A specific cyclic ADP-ribose antagonist inhibits cardiac excitation-contraction couplingS Rakovic, A Galione, G A Ashamu, et al.Cardiovascular Research|June 12, 2001
Nitric oxide does not modulate the hyperpolarization-activated current, I(f), in ventricular myocytes from spontaneously hypertensive ratsS M Bryant, C E Sears, L Rigg, et al.Psychosomatics|July 1, 1997
Consultation-liaison psychiatry and clinical ethics. Representative casesT PowellBasic Research in Cardiology|January 1, 1985
The isolation and characterization of calcium-tolerant myocytesT PowellBasic Research in Cardiology|January 1, 1985
Isolated adult heart cells. The development of a new experimental modelT PowellBasic Research in Cardiology|January 1, 1985
Electrophysiological properties of isolated ventricular myocytesT PowellPageof 70