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Plos One|August 17, 2017
D-cycloserine improves synaptic transmission in an animal model of Rett syndromeElisa S Na, Héctor De Jesús-Cortés, Arlene Martinez-Rivera, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 24, 2011
Cav1.2 L-type Ca²⁺ channels mediate cocaine-induced GluA1 trafficking in the nucleus accumbens, a long-term adaptation dependent on ventral tegmental area Ca(v)1.3 channelsKathryn Schierberl, Jin Hao, Thomas F Tropea, et al.
Reproduction, Fertility, and Development|March 3, 2005
Genetics and imaging to assess oocyte and preimplantation embryo healthC M Warner, J A Newmark, M Comiskey, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 2, 2017
Extinction of Contextual Cocaine Memories Requires Cav1.2 within D1R-Expressing Cells and Recruits Hippocampal Cav1.2-Dependent Signaling MechanismsCaitlin E Burgdorf, Kathryn C Schierberl, Anni S Lee, et al.
Biorxiv : the Preprint Server for Biology|April 25, 2024
Interneuron FGF13 regulates seizure susceptibility via a sodium channel-independent mechanismSusan Lin, Aravind R Gade, Hong-Gang Wang, et al.
Biorxiv : the Preprint Server for Biology|October 4, 2023
A Genetically Encoded Actuator Selectively Boosts L-type Calcium Channels in Diverse Physiological SettingsPedro J Del Rivero Morfin, Diego Scala Chavez, Srinidhi Jayaraman, et al.
American Journal of Human Genetics|November 13, 2010
Mutations in FLVCR1 cause posterior column ataxia and retinitis pigmentosaAnjali M Rajadhyaksha, Olivier Elemento, Erik G Puffenberger, et al.
Molecular Psychiatry|February 15, 2017
Enhancing VTA Cav1.3 L-type Ca2+ channel activity promotes cocaine and mood-related behaviors via overlapping AMPA receptor mechanisms in the nucleus accumbensA Martínez-Rivera, J Hao, T F Tropea, et al.
Molecular Psychiatry|April 26, 2020
Dopamine D1R-neuron cacna1c deficiency: a new model of extinction therapy-resistant post-traumatic stressCharlotte C Bavley, Zeeba D Kabir, Alexander P Walsh, et al.
Channels (Austin, Tex.)|June 27, 2025
Astrocytic abnormalities in brain-specific Cacna1c-deficient mice: Implications for BBB impairment in neuropsychiatric diseases associated with CACNA1C mutationsYeojung Koh, Maria Noterman-Soulinthavong, Anusha Bangalore, et al.
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