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Der Internist|March 8, 2005
[Therapy of organ failure in primary extracardiac diseases]S Reith, K WerdanNaunyn-Schmiedeberg'S Archives of Pharmacology|February 1, 1994
Tumor necrosis factor alpha decreases inositol phosphate formation and phosphatidylinositol-bisphosphate (PIP2) synthesis in rat cardiomyocytesC Reithmann, K WerdanMedizinische Klinik, Intensivmedizin Und Notfallmedizin|February 22, 2012
[Septic shock and septic cardiomyopathy]H Ebelt, K WerdanInfection|September 1, 1990
Cardiovascular parameters and scoring systems in the evaluation of response to therapy in sepsis and septic shockG Pilz, K WerdanNaunyn-Schmiedeberg'S Archives of Pharmacology|January 1, 1989
Noradrenaline-induced desensitization in cultured heart cells as a model for the defects of the adenylate cyclase system in severe heart failureC Reithmann, K WerdanBiomedizinische Technik. Biomedical Engineering|November 1, 1993
[Magnetomyography with the SQUID]M ReinckeHormone and Metabolic Research = Hormon- Und Stoffwechselforschung = Hormones Et Metabolisme|August 7, 1998
Mutations in adrenocortical tumorsM ReinckeNaunyn-Schmiedeberg'S Archives of Pharmacology|January 1, 1995
Chronic muscarinic cholinoceptor stimulation increases adenylyl cyclase responsiveness in rat cardiomyocytes by a decrease in the level of inhibitory G-protein alpha-subunitsC Reithmann, K WerdanEndocrinology and Metabolism Clinics of North America|March 25, 2000
Subclinical Cushing's syndromeM ReinckePageof 33