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Journal of Neural Transmission. Supplementum|November 29, 2002
The role of biological markers in the early and differential diagnosis of Alzheimer's diseaseA Kurz, M Riemenschneider, A Drzezga, et al.MMW Fortschritte Der Medizin|July 20, 2001
[Forgetfulness and light cognitive impairment. What can the physician still tolerate?]H Förstl, H Bickel, N Lautenschlager, et al.Dementia and Geriatric Cognitive Disorders|October 13, 2001
Primary degenerative mild cognitive impairment: study population, clinical, brain imaging and biochemical findingsN T Lautenschlager, M Riemenschneider, A Drzezga, et al.Der Nervenarzt|January 15, 2004
[Mild cognitive disorder. Questions of definition, diagnosis, prognosis and therapy]A Kurz, J Diehl, M Riemenschneider, et al.Journal of Neurology, Neurosurgery, and Psychiatry|April 24, 2002
Apolipoprotein E polymorphism in German patients with frontotemporal degenerationM Riemenschneider, J Diehl, U Müller, et al.Neuroscience Letters|July 19, 1996
Cerebrospinal protein tau is elevated in early Alzheimer's diseaseM Riemenschneider, K Buch, M Schmolke, et al.Multiple Sclerosis (Houndmills, Basingstoke, England)|July 3, 2020
Time matters: Early-phase multiple sclerosis is accompanied by considerable impairments across multiple domainsC Thrue, M Riemenschneider, L G Hvid, et al.Neurobiology of Aging|June 24, 2004
Cerebral metabolic patterns at early stages of frontotemporal dementia and semantic dementia. A PET studyJ Diehl, T Grimmer, A Drzezga, et al.The Journals of Gerontology. Series A, Biological Sciences and Medical Sciences|January 20, 2010
The cathepsin D (224C/T) polymorphism confers an increased risk to develop Alzheimer's disease in menO Albayrak, A Tirniceriu, M Riemenschneider, et al.Archives of Neurology|November 16, 2002
Cerebrospinal fluid tau and beta-amyloid 42 proteins identify Alzheimer disease in subjects with mild cognitive impairmentM Riemenschneider, N Lautenschlager, S Wagenpfeil, et al.Pageof 5