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FEBS Letters|January 27, 1992
Ca(2+)-induced Ca2+ release in insulin-secreting cellsM S Islam, P Rorsman, P O BerggrenFEBS Letters|August 19, 1991
Interaction with the inositol 1,4,5-trisphosphate receptor promotes Ca2+ sequestration in permeabilised insulin-secreting cellsM S Islam, T Nilsson, P Rorsman, et al.The American Journal of Physiology|December 1, 1986
Voltage-activated Na+ currents and their suppression by phorbol ester in clonal insulin-producing RINm5F cellsP Rorsman, P Arkhammar, P O BerggrenThe Biochemical Journal|February 15, 1982
Manganese accumulation in pancreatic beta-cells and its stimulation by glucoseP Rorsman, P O Berggren, B HellmanFEBS Letters|October 7, 1985
Direct measurements of increased free cytoplasmic Ca2+ in mouse pancreatic beta-cells following stimulation by hypoglycemic sulfonylureasH Abrahamsson, P O Berggren, P RorsmanBioscience Reports|April 1, 1986
Direct evidence for opposite effects of D-glucose and D-glyceraldehyde on cytoplasmic pH of mouse pancreatic beta-cellsP Arkhammar, P O Berggren, P RorsmanThe Biochemical Journal|July 15, 1993
Mobilization of Ca2+ by thapsigargin and 2,5-di-(t-butyl)-1,4-benzohydroquinone in permeabilized insulin-secreting RINm5F cells: evidence for separate uptake and release compartments in inositol 1,4,5-trisphosphate-sensitive Ca2+ poolM S Islam, P O BerggrenThe Journal of Biological Chemistry|January 15, 1989
Suppression of insulin release by galanin and somatostatin is mediated by a G-protein. An effect involving repolarization and reduction in cytoplasmic free Ca2+ concentrationT Nilsson, P Arkhammar, P Rorsman, et al.Bioscience Reports|October 1, 1983
Reduction of the cytosolic calcium activity in clonal insulin-releasing cells exposed to glucoseP Rorsman, P O Berggren, E Gylfe, et al.Pageof 72