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M S Sheikh

Showing results (51-60 of 64) with videos related to

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Cancer Research|November 1, 1995
Estradiol regulation of the human retinoic acid receptor alpha gene in human breast carcinoma cells is mediated via an imperfect half-palindromic estrogen response element and Sp1 motifsA K Rishi, Z M Shao, R G Baumann, et al.
Cancer Research|February 15, 1996
An abnormality in the p53 pathway following gamma-irradiation in many wild-type p53 human melanoma linesI Bae, M L Smith, M S Sheikh, et al.
Cancer Research|May 1, 1998
p53-dependent and -independent regulation of the death receptor KILLER/DR5 gene expression in response to genotoxic stress and tumor necrosis factor alphaM S Sheikh, T F Burns, Y Huang, et al.
Experimental Cell Research|May 1, 1995
Thyroid hormone enhancement of estradiol stimulation of breast carcinoma proliferationZ M Shao, M S Sheikh, A K Rishi, et al.
Cancer Research|September 25, 1992
IGFBP-3 gene expression and estrogen receptor status in human breast carcinomaZ M Shao, M S Sheikh, J V Ordonez, et al.
The Journal of Biological Chemistry|August 26, 1994
Retinoid-resistant estrogen receptor-negative human breast carcinoma cells transfected with retinoic acid receptor-alpha acquire sensitivity to growth inhibition by retinoidsM S Sheikh, Z M Shao, X S Li, et al.
Oncogene|August 3, 1995
p53 independent G0/G1 arrest and apoptosis induced by a novel retinoid in human breast cancer cellsZ M Shao, M I Dawson, X S Li, et al.
Journal of Cellular Physiology|December 1, 1995
Retinoic acid nuclear receptor beta inhibits breast carcinoma anchorage independent growthX S Li, Z M Shao, M S Sheikh, et al.
Carcinogenesis|October 1, 1995
N-(4-hydroxyphenyl)retinamide (4-HPR)-mediated biological actions involve retinoid receptor-independent pathways in human breast carcinomaM S Sheikh, Z M Shao, X S Li, et al.
Oncogene|August 6, 1999
The antiapoptotic decoy receptor TRID/TRAIL-R3 is a p53-regulated DNA damage-inducible gene that is overexpressed in primary tumors of the gastrointestinal tractM S Sheikh, Y Huang, E A Fernandez-Salas, et al.
Pageof 7

Showing results (51-60 of 64) with videos related to

Sort By:
Pageof 7
Cancer Research|November 1, 1995
Estradiol regulation of the human retinoic acid receptor alpha gene in human breast carcinoma cells is mediated via an imperfect half-palindromic estrogen response element and Sp1 motifsA K Rishi, Z M Shao, R G Baumann, et al.
Cancer Research|February 15, 1996
An abnormality in the p53 pathway following gamma-irradiation in many wild-type p53 human melanoma linesI Bae, M L Smith, M S Sheikh, et al.
Cancer Research|May 1, 1998
p53-dependent and -independent regulation of the death receptor KILLER/DR5 gene expression in response to genotoxic stress and tumor necrosis factor alphaM S Sheikh, T F Burns, Y Huang, et al.
Experimental Cell Research|May 1, 1995
Thyroid hormone enhancement of estradiol stimulation of breast carcinoma proliferationZ M Shao, M S Sheikh, A K Rishi, et al.
Cancer Research|September 25, 1992
IGFBP-3 gene expression and estrogen receptor status in human breast carcinomaZ M Shao, M S Sheikh, J V Ordonez, et al.
The Journal of Biological Chemistry|August 26, 1994
Retinoid-resistant estrogen receptor-negative human breast carcinoma cells transfected with retinoic acid receptor-alpha acquire sensitivity to growth inhibition by retinoidsM S Sheikh, Z M Shao, X S Li, et al.
Oncogene|August 3, 1995
p53 independent G0/G1 arrest and apoptosis induced by a novel retinoid in human breast cancer cellsZ M Shao, M I Dawson, X S Li, et al.
Journal of Cellular Physiology|December 1, 1995
Retinoic acid nuclear receptor beta inhibits breast carcinoma anchorage independent growthX S Li, Z M Shao, M S Sheikh, et al.
Carcinogenesis|October 1, 1995
N-(4-hydroxyphenyl)retinamide (4-HPR)-mediated biological actions involve retinoid receptor-independent pathways in human breast carcinomaM S Sheikh, Z M Shao, X S Li, et al.
Oncogene|August 6, 1999
The antiapoptotic decoy receptor TRID/TRAIL-R3 is a p53-regulated DNA damage-inducible gene that is overexpressed in primary tumors of the gastrointestinal tractM S Sheikh, Y Huang, E A Fernandez-Salas, et al.
Pageof 7