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The Journal of Biological Chemistry|November 29, 2018
Targeting BCL-xL improves the efficacy of bromodomain and extra-terminal protein inhibitors in triple-negative breast cancer by eliciting the death of senescent cellsSylvia S Gayle, Jennifer M Sahni, Bryan M Webb, et al.
Brain Pathology (Zurich, Switzerland)|July 1, 1997
Neuropathogenesis of chimeric simian/human immunodeficiency virus infection in pig-tailed and rhesus macaquesR Raghavan, E B Stephens, S V Joag, et al.
Leukemia|September 29, 2016
Pharmacogenomics and chemical library screens reveal a novel SCFSKP2 inhibitor that overcomes Bortezomib resistance in multiple myelomaE Malek, M A Y Abdel-Malek, S Jagannathan, et al.
Virology|May 12, 1997
A cell-free stock of simian-human immunodeficiency virus that causes AIDS in pig-tailed macaques has a limited number of amino acid substitutions in both SIVmac and HIV-1 regions of the genome and has offered cytotropismE B Stephens, S Mukherjee, M Sahni, et al.
Virology|July 27, 1999
Derivation and biological characterization of a molecular clone of SHIV(KU-2) that causes AIDS, neurological disease, and renal disease in rhesus macaquesZ Q Liu, S Muhkerjee, M Sahni, et al.
Cell|December 28, 2021
EMSY inhibits homologous recombination repair and the interferon response, promoting lung cancer immune evasionAntonio Marzio, Emma Kurz, Jennifer M Sahni, et al.
The Journal of Biological Chemistry|September 22, 2016
Bromodomain and Extraterminal Protein Inhibition Blocks Growth of Triple-negative Breast Cancers through the Suppression of Aurora KinasesJennifer M Sahni, Sylvia S Gayle, Kristen L Weber Bonk, et al.
Cancer Research|February 15, 2020
LIN9 and NEK2 Are Core Regulators of Mitotic Fidelity That Can Be Therapeutically Targeted to Overcome Taxane ResistanceMelyssa S Roberts, Jennifer M Sahni, Morgan S Schrock, et al.
Cancer Research|August 16, 2017
Mitotic Vulnerability in Triple-Negative Breast Cancer Associated with LIN9 Is Targetable with BET InhibitorsJennifer M Sahni, Sylvia S Gayle, Bryan M Webb, et al.
Cancer Research|May 12, 2025
Systemic Inactivation of TREX1 Induces Selective Inflammation of the Tumor Microenvironment and Invigorated T Cell-Mediated Tumor ControlEmilija Marinkovic, Minyi Chen, Nadja Schubert, et al.
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