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Nucleic Acids Research|February 25, 1991
Non-leucine residues in the leucine repeats of Fos and Jun contribute to the stability and determine the specificity of dimerizationM Schuermann, J B Hunter, G Hennig, et al.
Genes & Development|July 1, 1991
Alternative splicing of fosB transcripts results in differentially expressed mRNAs encoding functionally antagonistic proteinsD Mumberg, F C Lucibello, M Schuermann, et al.
The Journal of Biological Chemistry|March 4, 1994
T1 is a c-Fos- and FosB-responsive gene which is induced by growth factors through multiple signal transduction pathwaysM B Kalousek, T Trüb, M Schuermann, et al.
Nature|April 13, 1989
Two functionally different regions in Fos are required for the sequence-specific DNA interaction of the Fos/Jun protein complexM Neuberg, M Schuermann, J B Hunter, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|May 14, 1998
Frequent detection of ras and p53 mutations in brush cytology samples from lung cancer patients by a restriction fragment length polymorphism-based "enriched PCR" techniqueM Behn, S Qun, W Pankow, et al.
European Journal of Cancer (Oxford, England : 1990)|October 26, 2001
Axl receptor tyrosine kinase expression in human lung cancer cell lines correlates with cellular adhesionA Wimmel, D Glitz, A Kraus, et al.
The Journal of Pathology|January 21, 2000
Facilitated detection of oncogene mutations from exfoliated tissue material by a PNA-mediated 'enriched PCR' protocolM Behn, C Thiede, A Neubauer, et al.
Journal of Virology|March 1, 1989
A putative int domain for mouse mammary tumor virus on mouse chromosome 7 is a 5' extension of int-2G Peters, S Brookes, M Placzek, et al.
Journal of Cancer Research and Clinical Oncology|February 24, 2001
Transition from suspension to adherent growth is accompanied by tissue factor expression and matrix metalloproteinase secretion in a small cell lung cancer cell lineU Salge, R Seitz, A Wimmel, et al.
Journal of Clinical Oncology : Official Journal of the American Society of Clinical Oncology|September 14, 2000
Differential frequencies of p16(INK4a) promoter hypermethylation, p53 mutation, and K-ras mutation in exfoliative material mark the development of lung cancer in symptomatic chronic smokersM Kersting, C Friedl, A Kraus, et al.
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