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M Strittmatter

Showing results (21-30 of 326) with videos related to

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Current Opinion in Neurology|October 22, 2023
Development of neural repair therapy for chronic spinal cord trauma: soluble Nogo receptor decoy from discovery to clinical trialElisa M Howard, Stephen M Strittmatter
The Journal of Biological Chemistry|June 22, 2016
Oligomers of Amyloid β Prevent Physiological Activation of the Cellular Prion Protein-Metabotropic Glutamate Receptor 5 Complex by Glutamate in Alzheimer DiseaseLaura T Haas, Stephen M Strittmatter
Biochemical and Biophysical Research Communications|September 19, 2016
Cellular prion protein as a receptor for amyloid-β oligomers in Alzheimer's diseaseSantiago V Salazar, Stephen M Strittmatter
Journal of Neurochemistry|December 31, 1997
Brain CRMP forms heterotetramers similar to liver dihydropyrimidinaseL H Wang, S M Strittmatter
Current Opinion in Cell Biology|September 7, 2001
Semaphorin-mediated axonal guidance via Rho-related G proteinsB P Liu, S M Strittmatter
Archives of Neurology|November 11, 2009
Cellular prion protein mediates the toxicity of beta-amyloid oligomers: implications for Alzheimer diseaseHaakon B Nygaard, Stephen M Strittmatter
International MS Journal|September 11, 2008
Recurrent monofocal demyelinating diseaseP Jung, M Strittmatter, H Steinmetz
Science (New York, N.Y.)|November 27, 2010
Neuroscience. Lynx for braking plasticityMichael J Higley, Stephen M Strittmatter
Advances in Pharmacology (San Diego, Calif.)|February 8, 2018
Synaptotoxic Signaling by Amyloid Beta Oligomers in Alzheimer's Disease Through Prion Protein and mGluR5A Harrison Brody, Stephen M Strittmatter
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 1, 1996
A family of rat CRMP genes is differentially expressed in the nervous systemL H Wang, S M Strittmatter
Pageof 33

Showing results (21-30 of 326) with videos related to

Sort By:
Pageof 33
Current Opinion in Neurology|October 22, 2023
Development of neural repair therapy for chronic spinal cord trauma: soluble Nogo receptor decoy from discovery to clinical trialElisa M Howard, Stephen M Strittmatter
The Journal of Biological Chemistry|June 22, 2016
Oligomers of Amyloid β Prevent Physiological Activation of the Cellular Prion Protein-Metabotropic Glutamate Receptor 5 Complex by Glutamate in Alzheimer DiseaseLaura T Haas, Stephen M Strittmatter
Biochemical and Biophysical Research Communications|September 19, 2016
Cellular prion protein as a receptor for amyloid-β oligomers in Alzheimer's diseaseSantiago V Salazar, Stephen M Strittmatter
Journal of Neurochemistry|December 31, 1997
Brain CRMP forms heterotetramers similar to liver dihydropyrimidinaseL H Wang, S M Strittmatter
Current Opinion in Cell Biology|September 7, 2001
Semaphorin-mediated axonal guidance via Rho-related G proteinsB P Liu, S M Strittmatter
Archives of Neurology|November 11, 2009
Cellular prion protein mediates the toxicity of beta-amyloid oligomers: implications for Alzheimer diseaseHaakon B Nygaard, Stephen M Strittmatter
International MS Journal|September 11, 2008
Recurrent monofocal demyelinating diseaseP Jung, M Strittmatter, H Steinmetz
Science (New York, N.Y.)|November 27, 2010
Neuroscience. Lynx for braking plasticityMichael J Higley, Stephen M Strittmatter
Advances in Pharmacology (San Diego, Calif.)|February 8, 2018
Synaptotoxic Signaling by Amyloid Beta Oligomers in Alzheimer's Disease Through Prion Protein and mGluR5A Harrison Brody, Stephen M Strittmatter
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 1, 1996
A family of rat CRMP genes is differentially expressed in the nervous systemL H Wang, S M Strittmatter
Pageof 33