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M T Moslen

Showing results (1-10 of 47) with videos related to

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Advances in Experimental Medicine and Biology|January 1, 1994
Reactive oxygen species in normal physiology, cell injury and phagocytosisM T Moslen
Toxicologic Pathology|January 1, 1984
Increased incidence of hepatic foci and nodules in rats given one or two doses of 1,2-dibromoethaneM T Moslen
Research Communications in Chemical Pathology and Pharmacology|January 1, 1985
Rapid, substrate-specific, and dose-dependent deactivation of liver cytosolic glutathione S-transferases in vivo by 1,1-dichloroethyleneM T Moslen, E S Reynolds
Mutation Research|May 1, 1988
Advantages of a combined testing protocol (CTP) with multiple endpoints: the reverse tier approachM T Moslen, M S Legator
Environmental Health Perspectives|December 1, 1977
Damage to hepatic cellular membranes by chlorinated olefins with emphasis on synergism and antagonismE S Reynolds, M T Moslen
Toxicology and Applied Pharmacology|September 1, 1993
Biliary excretion of marker solutes by rats with 1,1-dichloroethylene-induced bile canalicular injuryM T Moslen, M F Kanz
Toxicology and Applied Pharmacology|December 16, 1998
Decreased biliary secretion of proteins and phospholipids by rats with 1,1-dichloroethylene-induced bile canalicular injuryS H Woodard, M T Moslen
The Journal of Histochemistry and Cytochemistry : Official Journal of the Histochemistry Society|March 1, 1988
Localization of high benzaldehyde dehydrogenase activity in rat upper gastrointestinal tract mucosa: a quantitative histochemical studyP Chieco, P Normanni, M T Moslen
Toxicology and Applied Pharmacology|July 1, 1996
Calcium channel blockers induce thymic apoptosis in vivo in ratsA Balakumaran, G A Campbell, M T Moslen
Laboratory Investigation; a Journal of Technical Methods and Pathology|April 1, 1982
Histochemical evidence that plasma and mitochondrial membranes are primary foci of hepatocellular injury caused by 1,1-dichloroethyleneP Chieco, M T Moslen, E S Reynolds
Pageof 5

Showing results (1-10 of 47) with videos related to

Sort By:
Pageof 5
Advances in Experimental Medicine and Biology|January 1, 1994
Reactive oxygen species in normal physiology, cell injury and phagocytosisM T Moslen
Toxicologic Pathology|January 1, 1984
Increased incidence of hepatic foci and nodules in rats given one or two doses of 1,2-dibromoethaneM T Moslen
Research Communications in Chemical Pathology and Pharmacology|January 1, 1985
Rapid, substrate-specific, and dose-dependent deactivation of liver cytosolic glutathione S-transferases in vivo by 1,1-dichloroethyleneM T Moslen, E S Reynolds
Mutation Research|May 1, 1988
Advantages of a combined testing protocol (CTP) with multiple endpoints: the reverse tier approachM T Moslen, M S Legator
Environmental Health Perspectives|December 1, 1977
Damage to hepatic cellular membranes by chlorinated olefins with emphasis on synergism and antagonismE S Reynolds, M T Moslen
Toxicology and Applied Pharmacology|September 1, 1993
Biliary excretion of marker solutes by rats with 1,1-dichloroethylene-induced bile canalicular injuryM T Moslen, M F Kanz
Toxicology and Applied Pharmacology|December 16, 1998
Decreased biliary secretion of proteins and phospholipids by rats with 1,1-dichloroethylene-induced bile canalicular injuryS H Woodard, M T Moslen
The Journal of Histochemistry and Cytochemistry : Official Journal of the Histochemistry Society|March 1, 1988
Localization of high benzaldehyde dehydrogenase activity in rat upper gastrointestinal tract mucosa: a quantitative histochemical studyP Chieco, P Normanni, M T Moslen
Toxicology and Applied Pharmacology|July 1, 1996
Calcium channel blockers induce thymic apoptosis in vivo in ratsA Balakumaran, G A Campbell, M T Moslen
Laboratory Investigation; a Journal of Technical Methods and Pathology|April 1, 1982
Histochemical evidence that plasma and mitochondrial membranes are primary foci of hepatocellular injury caused by 1,1-dichloroethyleneP Chieco, M T Moslen, E S Reynolds
Pageof 5