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FEBS Letters|November 23, 1992
Glucose decreases respiratory control ratio in EL-4 tumor cellsV L GabaiFEBS Letters|August 23, 1993
Inhibition of uncoupled respiration in tumor cells. A possible role of mitochondrial Ca2+ effluxV L GabaiThe Journal of Biological Chemistry|September 9, 2000
Suppression of stress kinase JNK is involved in HSP72-mediated protection of myogenic cells from transient energy deprivation. HSP72 alleviates the stewss-induced inhibition of JNK dephosphorylationV L Gabai, A B Meriin, J A Yaglom, et al.The Journal of Biological Chemistry|July 18, 1997
Hsp70 prevents activation of stress kinases. A novel pathway of cellular thermotoleranceV L Gabai, A B Meriin, D D Mosser, et al.Molecular and Cellular Biology|March 19, 1999
Protein-damaging stresses activate c-Jun N-terminal kinase via inhibition of its dephosphorylation: a novel pathway controlled by HSP72A B Meriin, J A Yaglom, V L Gabai, et al.Molecular and Cellular Biology|August 25, 2000
Hsp72-mediated suppression of c-Jun N-terminal kinase is implicated in development of tolerance to caspase-independent cell deathV L Gabai, J A Yaglom, V Volloch, et al.Experimental Cell Research|March 1, 1995
Heat shock-induced accumulation of 70-kDa stress protein (HSP70) can protect ATP-depleted tumor cells from necrosisA E Kabakov, V L GabaiTrends in Cell Biology|June 1, 1994
Heat-shock proteins maintain the viability of ATP-deprived cells: what is the mechanism?A E Kabakov, V L GabaiExperientia|August 15, 1993
Protein aggregation as primary and characteristic cell reaction to various stressesA E Kabakov, V L GabaiFEBS Letters|August 2, 1993
Rise in heat-shock protein level confers tolerance to energy deprivationV L Gabai, A E KabakovPageof 5