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The New England Journal of Medicine|September 10, 2010
Persistent malignant stem cells in del(5q) myelodysplasia in remissionRamin Tehranchi, Petter S Woll, Kristina Anderson, et al.Blood|February 8, 2013
miR-21 mediates hematopoietic suppression in MDS by activating TGF-β signalingTushar D Bhagat, Li Zhou, Lubomir Sokol, et al.Blood|August 4, 2012
STAT3 mutations unify the pathogenesis of chronic lymphoproliferative disorders of NK cells and T-cell large granular lymphocyte leukemiaAndres Jerez, Michael J Clemente, Hideki Makishima, et al.Blood|May 4, 2012
Loss of heterozygosity in 7q myeloid disorders: clinical associations and genomic pathogenesisAndres Jerez, Yuka Sugimoto, Hideki Makishima, et al.Cancer Research|December 30, 2010
Reduced SMAD7 leads to overactivation of TGF-beta signaling in MDS that can be reversed by a specific inhibitor of TGF-beta receptor I kinaseLi Zhou, Christine McMahon, Tushar Bhagat, et al.Frontiers in Immunology|December 22, 2018
Inhibition of Human Dendritic Cell ER Stress Response Reduces T Cell Alloreactivity Yet Spares Donor Anti-tumor ImmunityBrian C Betts, Frederick L Locke, Elizabeth M Sagatys, et al.Pageof 7