Showing results (21-30 of 59) with videos related to
Sort By:
Pageof 6
Aging|May 2, 2021
The nuclear sirtuin SIRT6 protects the heart from developing aging-associated myocyte senescence and cardiac hypertrophyVinodkumar B Pillai, Sadhana Samant, Samantha Hund, et al.The Journal of Biological Chemistry|February 6, 2008
HDAC4 and PCAF bind to cardiac sarcomeres and play a role in regulating myofilament contractile activityMahesh P Gupta, Sadhana A Samant, Stephen H Smith, et al.American Journal of Physiology. Heart and Circulatory Physiology|September 18, 2004
Increased expression of poly(ADP-ribose) polymerase-1 contributes to caspase-independent myocyte cell death during heart failureJyothish B Pillai, Hyde M Russell, Jai Raman, et al.Scientific Reports|September 21, 2017
The histone deacetylase SIRT6 blocks myostatin expression and development of muscle atrophySadhana A Samant, Abhinav Kanwal, Vinodkumar B Pillai, et al.Cell Biochemistry and Biophysics|July 27, 2005
Mitogen-activated protein kinases (p38 and c-Jun NH2-terminal kinase) are differentially regulated during cardiac volume and pressure overload hypertrophySomkiat Sopontammarak, Assad Aliharoob, Catherina Ocampo, et al.The Journal of Biological Chemistry|March 29, 2003
Calcium/calmodulin-dependent protein kinase activates serum response factor transcription activity by its dissociation from histone deacetylase, HDAC4. Implications in cardiac muscle gene regulation during hypertrophyFrancesca J Davis, Madhu Gupta, Blanca Camoretti-Mercado, et al.The Journal of Biological Chemistry|April 26, 2015
Histone Deacetylase 3 (HDAC3)-dependent Reversible Lysine Acetylation of Cardiac Myosin Heavy Chain Isoforms Modulates Their Enzymatic and Motor ActivitySadhana A Samant, Vinodkumar B Pillai, Nagalingam R Sundaresan, et al.The Journal of Clinical Investigation|August 5, 2009
Sirt3 blocks the cardiac hypertrophic response by augmenting Foxo3a-dependent antioxidant defense mechanisms in miceNagalingam R Sundaresan, Madhu Gupta, Gene Kim, et al.American Journal of Physiology. Heart and Circulatory Physiology|March 15, 2002
Increased expression of alternatively spliced dominant-negative isoform of SRF in human failing heartsFrancesca J Davis, Madhu Gupta, Steven M Pogwizd, et al.American Journal of Physiology. Heart and Circulatory Physiology|April 25, 2006
Poly(ADP-ribose) polymerase-1-deficient mice are protected from angiotensin II-induced cardiac hypertrophyJyothish B Pillai, Madhu Gupta, Senthilkumar B Rajamohan, et al.Pageof 6