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Maike Krenz

Showing results (1-10 of 44) with videos related to

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Cellular Signalling|March 8, 2022
Friend or foe? Unraveling the complex roles of protein tyrosine phosphatases in cardiac disease and developmentMaike Krenz
Journal of Applied Physiology (Bethesda, Md. : 1985)|October 3, 2009
Conductance, admittance, and hypertonic saline: should we take ventricular volume measurements with a grain of salt?Maike Krenz
Journal of Molecular and Cellular Cardiology|September 16, 2017
Heterozygous deletion of AKT1 rescues cardiac contractility, but not hypertrophy, in a mouse model of Noonan Syndrome with Multiple LentiginesRajika Roy, Maike Krenz
Journal of the American College of Cardiology|December 21, 2004
Impact of beta-myosin heavy chain expression on cardiac function during stressMaike Krenz, Jeffrey Robbins
Journal of Molecular and Cellular Cardiology|November 2, 2011
Moderate ethanol ingestion and cardiovascular protection: from epidemiologic associations to cellular mechanismsMaike Krenz, Ronald J Korthuis
Circulation Research|September 17, 2005
Noonan syndrome mutation Q79R in Shp2 increases proliferation of valve primordia mesenchymal cells via extracellular signal-regulated kinase 1/2 signalingMaike Krenz, Katherine E Yutzey, Jeffrey Robbins
The Journal of Biological Chemistry|May 16, 2013
New approaches to prevent LEOPARD syndrome-associated cardiac hypertrophy by specifically targeting Shp2-dependent signalingChristine Schramm, Michelle A Edwards, Maike Krenz
Annals of the New York Academy of Sciences|June 21, 2002
The protective and anti-protective effects of ethanol in a myocardial infarct modelMaike Krenz, Michael V Cohen, James M Downey
Pathophysiology : the Official Journal of the International Society for Pathophysiology|March 10, 2004
Protective and anti-protective effects of acute ethanol exposure in myocardial ischemia/reperfusionMaike Krenz, Michael V Cohen, James M Downey
Journal of Applied Physiology (Bethesda, Md. : 1985)|November 1, 2014
The Q510E mutation in Shp2 perturbs heart valve development by increasing cell migrationMichelle A Edwards, Kathryn Crombie, Christine Schramm, et al.
Pageof 5

Showing results (1-10 of 44) with videos related to

Sort By:
Pageof 5
Cellular Signalling|March 8, 2022
Friend or foe? Unraveling the complex roles of protein tyrosine phosphatases in cardiac disease and developmentMaike Krenz
Journal of Applied Physiology (Bethesda, Md. : 1985)|October 3, 2009
Conductance, admittance, and hypertonic saline: should we take ventricular volume measurements with a grain of salt?Maike Krenz
Journal of Molecular and Cellular Cardiology|September 16, 2017
Heterozygous deletion of AKT1 rescues cardiac contractility, but not hypertrophy, in a mouse model of Noonan Syndrome with Multiple LentiginesRajika Roy, Maike Krenz
Journal of the American College of Cardiology|December 21, 2004
Impact of beta-myosin heavy chain expression on cardiac function during stressMaike Krenz, Jeffrey Robbins
Journal of Molecular and Cellular Cardiology|November 2, 2011
Moderate ethanol ingestion and cardiovascular protection: from epidemiologic associations to cellular mechanismsMaike Krenz, Ronald J Korthuis
Circulation Research|September 17, 2005
Noonan syndrome mutation Q79R in Shp2 increases proliferation of valve primordia mesenchymal cells via extracellular signal-regulated kinase 1/2 signalingMaike Krenz, Katherine E Yutzey, Jeffrey Robbins
The Journal of Biological Chemistry|May 16, 2013
New approaches to prevent LEOPARD syndrome-associated cardiac hypertrophy by specifically targeting Shp2-dependent signalingChristine Schramm, Michelle A Edwards, Maike Krenz
Annals of the New York Academy of Sciences|June 21, 2002
The protective and anti-protective effects of ethanol in a myocardial infarct modelMaike Krenz, Michael V Cohen, James M Downey
Pathophysiology : the Official Journal of the International Society for Pathophysiology|March 10, 2004
Protective and anti-protective effects of acute ethanol exposure in myocardial ischemia/reperfusionMaike Krenz, Michael V Cohen, James M Downey
Journal of Applied Physiology (Bethesda, Md. : 1985)|November 1, 2014
The Q510E mutation in Shp2 perturbs heart valve development by increasing cell migrationMichelle A Edwards, Kathryn Crombie, Christine Schramm, et al.
Pageof 5