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The Journal of General Physiology|December 23, 2019
An interaction between the III-IV linker and CTD in NaV1.5 confers regulation of inactivation by CaM and FHFAravind R Gade, Steven O Marx, Geoffrey S Pitt
The Journal of General Physiology|January 15, 2017
Calmodulin limits pathogenic Na+ channel persistent currentHaidun Yan, Chaojian Wang, Steven O Marx, et al.
Elife|October 1, 2014
FGF14 modulates resurgent sodium current in mouse cerebellar Purkinje neuronsHaidun Yan, Juan L Pablo, Chaojian Wang, et al.
Neuron|March 9, 2004
Identification of the components controlling inactivation of voltage-gated Ca2+ channelsJames Kim, Smita Ghosh, Deborah A Nunziato, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 22, 2008
Ca2+/CaM controls Ca2+-dependent inactivation of NMDA receptors by dimerizing the NR1 C terminiChaojian Wang, Hong-Gang Wang, Hui Xie, et al.
Nature Structural & Molecular Biology|March 8, 2005
Essential Ca(V)beta modulatory properties are AID-independentJanet M Maltez, Deborah A Nunziato, James Kim, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 5, 2016
Polarized localization of voltage-gated Na+ channels is regulated by concerted FGF13 and FGF14 actionJuan Lorenzo Pablo, Chaojian Wang, Matthew M Presby, et al.
Plos One|March 31, 2016
α1-Syntrophin Variant Identified in Drug-Induced Long QT Syndrome Increases Late Sodium CurrentJong-Il Choi, Chaojian Wang, Matthew J Thomas, et al.
Trends in Cardiovascular Medicine|August 8, 2012
Fibroblast growth factor homologous factors in the heart: a potential locus for cardiac arrhythmiasEric Q Wei, Adam S Barnett, Geoffrey S Pitt, et al.
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