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Molecular and Cellular Biochemistry|July 23, 2003
Lysophosphatidylcholine-induced myocardial damage is inhibited by pretreatment with poloxamer 188 in isolated rat heartMakino Watanabe, Takao Okada
Methods in Molecular Biology (Clifton, N.J.)|July 11, 2018
Langendorff Perfusion Method as an Ex Vivo Model to Evaluate Heart Function in RatsMakino Watanabe, Takao Okada
Experimental and Clinical Cardiology|July 31, 2009
Mechanisms of cardioprotective effects of magnesium on hypoxia-reoxygenation-induced injuryMakino Watanabe, Jinrong Wu, Shanshuang Li, et al.
The Journal of Physiological Sciences : JPS|April 25, 2012
Mitochondrial K+ channels are involved in ischemic postconditioning in rat heartsChunhong Jin, Jinrong Wu, Makino Watanabe, et al.
Journal of Pharmacological Sciences|January 22, 2022
Chronic magnesium deficiency causes reversible mitochondrial permeability transition pore opening and impairs hypoxia tolerance in the rat heartMakino Watanabe, Kyoko Nakamura, Megumi Kato, et al.
Experimental and Clinical Cardiology|July 25, 2008
Protective effects of ischemic postconditioning against hypoxia-reoxygenation injury and hydrogen peroxide-induced damage in isolated rat heartsShanshuang Li, Jinrong Wu, Makino Watanabe, et al.
The Journal of Physiological Sciences : JPS|April 3, 2009
Sevoflurane and nitrous oxide exert cardioprotective effects against hypoxia-reoxygenation injury in the isolated rat heartChunhong Jin, Seijiro Sonoda, Liu Fan, et al.
The Journal of Histochemistry and Cytochemistry : Official Journal of the Histochemistry Society|November 30, 2005
Alterations of phosphorylation state of connexin 43 during hypoxia and reoxygenation are associated with cardiac functionSatoshi Matsushita, Hidetake Kurihara, Makino Watanabe, et al.
Pediatrics International : Official Journal of the Japan Pediatric Society|May 26, 2010
Intracellular magnesium and tolerance against hypoxia in immature rat heartTomoyuki Sato, Makino Watanabe, Masahiko Kishiro, et al.
Histology and Histopathology|January 7, 2011
Inhibition of connexin43 dephosphorylation is involved in protective effects of diltiazem on cardiac function during hypoxic injurySatoshi Matsushita, Hidetake Kurihara, Makino Watanabe, et al.
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