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Biological Trace Element Research|April 14, 2020
Extracellular Zn2+-Dependent Amyloid-β1-42 Neurotoxicity in Alzheimer's Disease PathogenesisYuichi Sato, Mako Takiguchi, Haruna Tamano, et al.Biochemical and Biophysical Research Communications|May 16, 2019
Extracellular Zn2+-independently attenuated LTP by human amyloid β1-40 and rat amyloid β1-42Haruna Tamano, Mako Takiguchi, Ryota Shimaya, et al.Molecular Neurobiology|December 23, 2019
Preferential Neurodegeneration in the Dentate Gyrus by Amyloid β1-42-Induced Intracellular Zn2+Dysregulation and Its Defense StrategyHaruna Tamano, Mako Takiguchi, Yukino Tanaka, et al.Cellular and Molecular Neurobiology|June 1, 2019
CA1 LTP Attenuated by Corticosterone is Canceled by Effusol via Rescuing Intracellular Zn2+ DysregulationHaruna Tamano, Yuichi Sato, Mako Takiguchi, et al.Neuroscience Letters|March 4, 2024
Blockage of metallothionein synthesis via adrenaline β receptor activation invalidates dehydroeffusol-mediated prevention of amyloid β1-42 toxicityHaruna Tamano, Mako Takiguchi, Daichi Murakami, et al.Plant Foods for Human Nutrition (Dordrecht, Netherlands)|April 26, 2020
Dehydroeffusol Rescues Amyloid β25-35-Induced Spatial Working Memory DeficitToshiyuki Fukuda, Yuichi Sato, Mako Takiguchi, et al.Molecular Neurobiology|March 26, 2021
Dehydroeffusol Pprevents Amyloid β1-42-mediated Hippocampal Neurodegeneration via Reducing Intracellular Zn2+ ToxicityHaruna Tamano, Mako Takiguchi, Nana Saeki, et al.Pageof 1