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Journal of Molecular Medicine (Berlin, Germany)|April 3, 2017
Synovial cell production of IL-26 induces bone mineralization in spondyloarthritisLine Dam Heftdal, Thomas Andersen, Ditte Jæhger, et al.Arthritis Research & Therapy|February 5, 2014
Decreased plasma levels of soluble CD18 link leukocyte infiltration with disease activity in spondyloarthritisTue W Kragstrup, Babak Jalilian, Malene Hvid, et al.Frontiers in Immunology|August 10, 2017
Extracellular Vesicles Transfer the Receptor Programmed Death-1 in Rheumatoid ArthritisStinne R Greisen, Yan Yan, Aida S Hansen, et al.American Journal of Clinical and Experimental Immunology|July 25, 2022
T-cell immunoglobulin and mucin domain 3 is upregulated in rheumatoid arthritis, but insufficient in controlling inflammationCaecilie Skejoe, Aida S Hansen, Kristian Stengaard-Pedersen, et al.Scientific Reports|November 13, 2021
Phenotypic and functional characterization of synovial fluid-derived fibroblast-like synoviocytes in rheumatoid arthritisDitte Køster, Johanne Hovgaard Egedal, Søren Lomholt, et al.Rheumatology International|December 1, 2010
Increased plasma levels of IL-21 and IL-23 in spondyloarthritis are not associated with clinical and MRI findingsThomas Andersen, Tue Kruse Rasmussen, Malene Hvid, et al.Journal for Immunotherapy of Cancer|October 2, 2024
Gal-3 blocks the binding between PD-1 and pembrolizumabStinne Ravn Greisen, Mia Bendix, Morten Aagaard Nielsen, et al.Scandinavian Journal of Clinical and Laboratory Investigation|June 10, 2017
Soluble CD206 plasma levels in rheumatoid arthritis reflect decrease in disease activityLine Dam Heftdal, Kristian Stengaard-Pedersen, Lykke Midtbøll Ørnbjerg, et al.Arthritis Research & Therapy|September 25, 2014
CXCL13 predicts disease activity in early rheumatoid arthritis and could be an indicator of the therapeutic 'window of opportunity'Stinne Ravn Greisen, Karen Kræmmer Schelde, Tue Kruse Rasmussen, et al.Annals of the Rheumatic Diseases|February 10, 2026
Engaging the PD-1 pathway in systemic sclerosis attenuates inflammation-driven fibrosisMaithri Aspari, Voon Ong, Klaus Søndergaard, et al.Pageof 5