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Cellular Immunology|June 7, 2005
Ncf1 (p47phox) polymorphism determines oxidative burst and the severity of arthritis in rats and miceMalin Hultqvist, Rikard HolmdahlArthritis Research & Therapy|June 4, 2009
The value of animal models in predicting genetic susceptibility to complex diseases such as rheumatoid arthritisEmma Ahlqvist, Malin Hultqvist, Rikard HolmdahlAnnals of the Rheumatic Diseases|June 15, 2010
Rabeximod reduces arthritis severity in mice by decreasing activation of inflammatory cellsMalin Hultqvist, Kutty Selva Nandakumar, Ulf Björklund, et al.Antioxidants & Redox Signaling|December 24, 2013
Pharmacological Potential of NOX2 Agonists in Inflammatory ConditionsMalin Hultqvist, Peter Olofsson, Fredrik K Wallner, et al.The American Journal of Pathology|May 31, 2012
Enhancement of antibody-induced arthritis via Toll-like receptor 2 stimulation is regulated by granulocyte reactive oxygen speciesTiina Kelkka, Malin Hultqvist, Kutty Selva Nandakumar, et al.Antioxidants & Redox Signaling|May 28, 2016
A Reduction in Intracellular Reactive Oxygen Species Due to a Mutation in NCF4 Promotes Autoimmune Arthritis in MiceSusann Winter, Malin Hultqvist Hopkins, Frida Laulund, et al.Antioxidants & Redox Signaling|October 6, 2010
NOX2 complex-derived ROS as immune regulatorsOuti Sareila, Tiina Kelkka, Angela Pizzolla, et al.Trends in Immunology|April 10, 2009
The protective role of ROS in autoimmune diseaseMalin Hultqvist, Lina M Olsson, Kyra A Gelderman, et al.Proceedings of the National Academy of Sciences of the United States of America|August 16, 2006
T cell surface redox levels determine T cell reactivity and arthritis susceptibilityKyra A Gelderman, Malin Hultqvist, Jens Holmberg, et al.Journal of Immunology (Baltimore, Md. : 1950)|July 21, 2007
Lack of reactive oxygen species breaks T cell tolerance to collagen type II and allows development of arthritis in miceMalin Hultqvist, Johan Bäcklund, Kristin Bauer, et al.Pageof 4