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The Journal of General Virology
|
March 26, 2003
Molecular analysis of iatrogenic scrapie in Italy
Gianluigi Zanusso, Cristina Casalone, Pierluigi Acutis, et al.
The Journal of Biological Chemistry
|
June 30, 2023
Persistent ER stress causes GPI anchor deficit to convert a GPI-anchored prion protein into pro-PrP via the ATF6-miR449c-5p-PIGV axis
JingFeng Li, SaSa Li, ShuPei Yu, et al.
The Journal of Biological Chemistry
|
December 20, 2015
Glycosylphosphatidylinositol Anchor Modification Machinery Deficiency Is Responsible for the Formation of Pro-Prion Protein (PrP) in BxPC-3 Protein and Increases Cancer Cell Motility
Liheng Yang, Zhenxing Gao, Lipeng Hu, et al.
Journal of Alzheimer'S Disease : JAD
|
April 27, 2005
Characterization of the F198S prion protein mutation: enhanced glycosylation and defective refolding
Syed I A Zaidi, Sandra L Richardson, Sabina Capellari, et al.
The Biochemical Journal
|
December 26, 2006
Aggregation of prion protein with insertion mutations is proportional to the number of inserts
Shuiliang Yu, Shaoman Yin, Chaoyang Li, et al.
Plos One
|
September 23, 2016
Polyglutamine Tract Expansion Increases S-Nitrosylation of Huntingtin and Ataxin-1
Chun-Lun Ni, Divya Seth, Fabio Vasconcelos Fonseca, et al.
Neuroscience Letters
|
December 31, 2002
Anti-prion antibodies for prophylaxis following prion exposure in mice
Einar M Sigurdsson, Man-Sun Sy, Ruliang Li, et al.
The Journal of Biological Chemistry
|
March 27, 2016
Glycosylphosphatidylinositol anchor modification machinery deficiency is responsible for the formation of pro-prion protein (PrP) in BxPC-3 cells and increases cancer cell motility
Liheng Yang, Zhenxing Gao, Lipeng Hu, et al.
Biochemical and Biophysical Research Communications
|
December 25, 2019
Melanoma migration is promoted by prion protein via Akt-hsp27 signaling axis
Jingru Ke, Guiru Wu, Jie Zhang, et al.
Virologica Sinica
|
November 25, 2020
Tumor Necrosis Factor α Reduces SNAP29 Dependent Autolysosome Formation to Increase Prion Protein Level and Promote Tumor Cell Migration
Huan Li, Ren Wang, Ze Yu, et al.
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of 8
Search research articles
Search
Showing results (41-50 of 73) with videos related to
Sort By:
Page
of 8
The Journal of General Virology
|
March 26, 2003
Molecular analysis of iatrogenic scrapie in Italy
Gianluigi Zanusso, Cristina Casalone, Pierluigi Acutis, et al.
The Journal of Biological Chemistry
|
June 30, 2023
Persistent ER stress causes GPI anchor deficit to convert a GPI-anchored prion protein into pro-PrP via the ATF6-miR449c-5p-PIGV axis
JingFeng Li, SaSa Li, ShuPei Yu, et al.
The Journal of Biological Chemistry
|
December 20, 2015
Glycosylphosphatidylinositol Anchor Modification Machinery Deficiency Is Responsible for the Formation of Pro-Prion Protein (PrP) in BxPC-3 Protein and Increases Cancer Cell Motility
Liheng Yang, Zhenxing Gao, Lipeng Hu, et al.
Journal of Alzheimer'S Disease : JAD
|
April 27, 2005
Characterization of the F198S prion protein mutation: enhanced glycosylation and defective refolding
Syed I A Zaidi, Sandra L Richardson, Sabina Capellari, et al.
The Biochemical Journal
|
December 26, 2006
Aggregation of prion protein with insertion mutations is proportional to the number of inserts
Shuiliang Yu, Shaoman Yin, Chaoyang Li, et al.
Plos One
|
September 23, 2016
Polyglutamine Tract Expansion Increases S-Nitrosylation of Huntingtin and Ataxin-1
Chun-Lun Ni, Divya Seth, Fabio Vasconcelos Fonseca, et al.
Neuroscience Letters
|
December 31, 2002
Anti-prion antibodies for prophylaxis following prion exposure in mice
Einar M Sigurdsson, Man-Sun Sy, Ruliang Li, et al.
The Journal of Biological Chemistry
|
March 27, 2016
Glycosylphosphatidylinositol anchor modification machinery deficiency is responsible for the formation of pro-prion protein (PrP) in BxPC-3 cells and increases cancer cell motility
Liheng Yang, Zhenxing Gao, Lipeng Hu, et al.
Biochemical and Biophysical Research Communications
|
December 25, 2019
Melanoma migration is promoted by prion protein via Akt-hsp27 signaling axis
Jingru Ke, Guiru Wu, Jie Zhang, et al.
Virologica Sinica
|
November 25, 2020
Tumor Necrosis Factor α Reduces SNAP29 Dependent Autolysosome Formation to Increase Prion Protein Level and Promote Tumor Cell Migration
Huan Li, Ren Wang, Ze Yu, et al.
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of 8