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Frontiers in Cardiovascular Medicine|October 12, 2023
Myocardial oxidative stress is increased in early reperfusion, but systemic antioxidative therapy does not prevent ischemia-reperfusion arrhythmias in pigsMarie Haugsten Hansen, Mani Sadredini, Almira Hasic, et al.
Journal of Molecular and Cellular Cardiology|December 17, 2022
CaMKII and reactive oxygen species contribute to early reperfusion arrhythmias, but oxidation of CaMKIIδ at methionines 281/282 is not a determining factorMarie Haugsten Hansen, Mani Sadredini, Almira Hasic, et al.
Physiological Reports|September 24, 2021
The oxidation-resistant CaMKII-MM281/282VV mutation does not prevent arrhythmias in CPVT1Mani Sadredini, Ravinea Manotheepan, Stephan E Lehnart, et al.
American Journal of Physiology. Heart and Circulatory Physiology|July 16, 2021
CaMKII inhibition has dual effects on spontaneous Ca2+ release and Ca2+ alternans in ventricular cardiomyocytes from mice with a gain-of-function RyR2 mutationMani Sadredini, Marie Haugsten Hansen, Michael Frisk, et al.
Cardiovascular Research|May 11, 2016
Exercise training prevents ventricular tachycardia in CPVT1 due to reduced CaMKII-dependent arrhythmogenic Ca2+ releaseRavinea Manotheepan, Tore K Danielsen, Mani Sadredini, et al.
American Journal of Physiology. Heart and Circulatory Physiology|April 28, 2023
Beneficial effects of exercise initiated before development of hypertrophic cardiomyopathy in genotype-positive miceKristine Andreassen, Chloe Rixon, Marie H Hansen, et al.
Frontiers in Cardiovascular Medicine|February 11, 2021
Exercise Training Stabilizes RyR2-Dependent Ca2+ Release in Post-infarction Heart FailureTore Kristian Danielsen, Mani Sadredini, Ravinea Manotheepan, et al.
Circulation. Arrhythmia and Electrophysiology|April 5, 2019
Secretoneurin Is an Endogenous Calcium/Calmodulin-Dependent Protein Kinase II Inhibitor That Attenuates Ca2+-Dependent ArrhythmiaAnett H Ottesen, Cathrine R Carlson, Olav Søvik Eken, et al.
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