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Neuropharmacology|October 17, 2018
Characterisation of the mechanisms underlying the special sensitivity of the CA2 hippocampal area to adenosine receptor antagonistsMaría-Dolores Muñoz, José M SolísJournal of Neurochemistry|August 12, 2005
Chronic hyperammonemia in vivo impairs long-term potentiation in hippocampus by altering activation of cyclic GMP-dependent-protein kinase and of phosphodiesterase 5Pilar Monfort, María-Dolores Muñoz, Vicente FelipoMetabolic Brain Disease|December 31, 2005
Molecular mechanisms of the alterations in NMDA receptor-dependent long-term potentiation in hyperammonemiaPilar Monfort, María-Dolores Muñoz, Vicente FelipoNeurobiology of Disease|January 31, 2004
Hyperammonemia impairs long-term potentiation in hippocampus by altering the modulation of cGMP-degrading phosphodiesterase by protein kinase GPilar Monfort, María-Dolores Muñoz, Vicente FelipoThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 27, 2006
Involvement of the CA3-CA1 synapse in the acquisition of associative learning in behaving miceAgnès Gruart, María Dolores Muñoz, José M Delgado-GarcíaThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 27, 2002
Long-term potentiation in hippocampus involves sequential activation of soluble guanylate cyclase, cGMP-dependent protein kinase, and cGMP-degrading phosphodiesterasePilar Monfort, María-Dolores Muñoz, Elena Kosenko, et al.Metabolic Brain Disease|February 27, 2003
Effects of hyperammonemia and liver failure on glutamatergic neurotransmissionPilar Monfort, María-Dolores Muñoz, Amina ElAyadi, et al.Translational Stroke Research|September 27, 2023
Identification of Non-excitatory Amino Acids and Transporters Mediating the Irreversible Synaptic Silencing After HypoxiaIris Álvarez-Merz, María-Dolores Muñoz, Jesús M Hernández-Guijo, et al.Amino Acids|January 25, 2016
Taurine content in different brain structures during ageing: effect on hippocampal synaptic plasticityLuz M Suárez, María-Dolores Muñoz, Rafael Martín Del Río, et al.Neurochemistry International|August 18, 2004
Sequential activation of soluble guanylate cyclase, protein kinase G and cGMP-degrading phosphodiesterase is necessary for proper induction of long-term potentiation in CA1 of hippocampus. Alterations in hyperammonemiaPilar Monfort, María-Dolores Muñoz, Elena Kosenko, et al.Pageof 2