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Annals of Medicine|July 20, 2005
Atherosclerosis and innate immune signalingMarc A Laberge, Kathryn J Moore, Mason W Freeman
Arteriosclerosis, Thrombosis, and Vascular Biology|May 27, 2006
Scavenger receptors in atherosclerosis: beyond lipid uptakeKathryn J Moore, Mason W Freeman
Drug Discovery Today. Therapeutic Strategies|May 12, 2009
Targeting Innate Immunity for CV BenefitKathryn J Moore, Mason W Freeman
Journal of Molecular Medicine (Berlin, Germany)|May 22, 2002
Nuclear hormone receptors and cholesterol trafficking: the orphans find a new homeMichael L Fitzgerald, Kathryn J Moore, Mason W Freeman
The Journal of Biological Chemistry|September 20, 2002
A CD36-initiated signaling cascade mediates inflammatory effects of beta-amyloidKathryn J Moore, Joseph El Khoury, Lea A Medeiros, et al.
The Journal of Experimental Medicine|June 11, 2003
CD36 mediates the innate host response to beta-amyloidJoseph B El Khoury, Kathryn J Moore, Terry K Means, et al.
The Journal of Clinical Investigation|August 3, 2005
Loss of receptor-mediated lipid uptake via scavenger receptor A or CD36 pathways does not ameliorate atherosclerosis in hyperlipidemic miceKathryn J Moore, Vidya V Kunjathoor, Stephanie L Koehn, et al.
The Journal of Biological Chemistry|October 12, 2002
Scavenger receptors class A-I/II and CD36 are the principal receptors responsible for the uptake of modified low density lipoprotein leading to lipid loading in macrophagesVidya V Kunjathoor, Maria Febbraio, Eugene A Podrez, et al.
The Journal of Biological Chemistry|November 20, 2004
Abca7 null mice retain normal macrophage phosphatidylcholine and cholesterol efflux activity despite alterations in adipose mass and serum cholesterol levelsWoojin Scott Kim, Michael L Fitzgerald, Kihwa Kang, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|October 25, 2008
Loss of SR-A and CD36 activity reduces atherosclerotic lesion complexity without abrogating foam cell formation in hyperlipidemic miceJennifer J Manning-Tobin, Kathryn J Moore, Tracie A Seimon, et al.
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