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Marc Hafner

Showing results (41-50 of 53) with videos related to

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Science Translational Medicine|September 21, 2022
Giredestrant reverses progesterone hypersensitivity driven by estrogen receptor mutations in breast cancerJackson Liang, Ellen Rei Ingalla, Xiaosai Yao, et al.
Cell Chemical Biology|May 20, 2026
Giredestrant immobilizes the estrogen receptor to exert potent antitumor efficacy against ER-active breast cancersWei Zhou, Musaddeque Ahmed, Jane Guan, et al.
Cell|July 30, 2019
Therapeutic Ligands Antagonize Estrogen Receptor Function by Impairing Its MobilityJane Guan, Wei Zhou, Marc Hafner, et al.
Cancer Discovery|September 21, 2021
RTK-Dependent Inducible Degradation of Mutant PI3Kα Drives GDC-0077 (Inavolisib) EfficacyKyung W Song, Kyle A Edgar, Emily J Hanan, et al.
Nature Communications|August 2, 2025
Epiregulon: Single-cell transcription factor activity inference to predict drug response and drivers of cell statesTomasz Włodarczyk, Aaron Lun, Diana Wu, et al.
Journal of Medicinal Chemistry|June 22, 2026
Utilizing Molecular Dynamics and Mechanistic Pharmacokinetic Studies in the Design of Selective CDK2 InhibitorsVishal A Verma, Jessica M Grandner, Brendan T Parr, et al.
Bioorganic & Medicinal Chemistry Letters|July 17, 2019
Design of a brain-penetrant CDK4/6 inhibitor for glioblastomaSarah M Bronner, Karl A Merrick, Jeremy Murray, et al.
Nature Cancer|June 5, 2023
An allosteric pan-TEAD inhibitor blocks oncogenic YAP/TAZ signaling and overcomes KRAS G12C inhibitor resistanceThijs J Hagenbeek, Jason R Zbieg, Marc Hafner, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|October 27, 2025
PI3Kα Inhibitor and Degrader Inavolisib Can Co-opt FGFR2 to Enhance Responses in Patients with PIK3CA-Mutated Solid Tumors and in Preclinical ModelsDejan Juric, Kyung Song, Radia M Johnson, et al.
Nature Communications|June 27, 2026
Covalent pan-TEAD inhibitors block YAP activity and demonstrate brain penetrance in a Hippo-dependent cancer modelThijs J Hagenbeek, Jason Zbieg, Russell Smith, et al.
Pageof 6

Showing results (41-50 of 53) with videos related to

Sort By:
Pageof 6
Science Translational Medicine|September 21, 2022
Giredestrant reverses progesterone hypersensitivity driven by estrogen receptor mutations in breast cancerJackson Liang, Ellen Rei Ingalla, Xiaosai Yao, et al.
Cell Chemical Biology|May 20, 2026
Giredestrant immobilizes the estrogen receptor to exert potent antitumor efficacy against ER-active breast cancersWei Zhou, Musaddeque Ahmed, Jane Guan, et al.
Cell|July 30, 2019
Therapeutic Ligands Antagonize Estrogen Receptor Function by Impairing Its MobilityJane Guan, Wei Zhou, Marc Hafner, et al.
Cancer Discovery|September 21, 2021
RTK-Dependent Inducible Degradation of Mutant PI3Kα Drives GDC-0077 (Inavolisib) EfficacyKyung W Song, Kyle A Edgar, Emily J Hanan, et al.
Nature Communications|August 2, 2025
Epiregulon: Single-cell transcription factor activity inference to predict drug response and drivers of cell statesTomasz Włodarczyk, Aaron Lun, Diana Wu, et al.
Journal of Medicinal Chemistry|June 22, 2026
Utilizing Molecular Dynamics and Mechanistic Pharmacokinetic Studies in the Design of Selective CDK2 InhibitorsVishal A Verma, Jessica M Grandner, Brendan T Parr, et al.
Bioorganic & Medicinal Chemistry Letters|July 17, 2019
Design of a brain-penetrant CDK4/6 inhibitor for glioblastomaSarah M Bronner, Karl A Merrick, Jeremy Murray, et al.
Nature Cancer|June 5, 2023
An allosteric pan-TEAD inhibitor blocks oncogenic YAP/TAZ signaling and overcomes KRAS G12C inhibitor resistanceThijs J Hagenbeek, Jason R Zbieg, Marc Hafner, et al.
Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|October 27, 2025
PI3Kα Inhibitor and Degrader Inavolisib Can Co-opt FGFR2 to Enhance Responses in Patients with PIK3CA-Mutated Solid Tumors and in Preclinical ModelsDejan Juric, Kyung Song, Radia M Johnson, et al.
Nature Communications|June 27, 2026
Covalent pan-TEAD inhibitors block YAP activity and demonstrate brain penetrance in a Hippo-dependent cancer modelThijs J Hagenbeek, Jason Zbieg, Russell Smith, et al.
Pageof 6