Showing results (21-30 of 51) with videos related to
Sort By:
Pageof 6
Blood|October 1, 2008
Critical role of scavenger receptor-BI-expressing bone marrow-derived endothelial progenitor cells in the attenuation of allograft vasculopathy after human apo A-I transferYingmei Feng, Miranda van Eck, Eline Van Craeyveld, et al.The Journal of Clinical Investigation|March 11, 2009
Membrane-anchored uPAR regulates the proliferation, marrow pool size, engraftment, and mobilization of mouse hematopoietic stem/progenitor cellsMarc Tjwa, Nicolai Sidenius, Rute Moura, et al.Diabetes|November 26, 2013
Diabetes causes bone marrow autonomic neuropathy and impairs stem cell mobilization via dysregulated p66Shc and Sirt1Mattia Albiero, Nicol Poncina, Marc Tjwa, et al.Blood|December 8, 2009
Malignant cells fuel tumor growth by educating infiltrating leukocytes to produce the mitogen Gas6Sonja Loges, Thomas Schmidt, Marc Tjwa, et al.Journal of Molecular and Cellular Cardiology|July 4, 2006
A SERCA2 pump with an increased Ca2+ affinity can lead to severe cardiac hypertrophy, stress intolerance and reduced life spanPeter Vangheluwe, Marc Tjwa, An Van Den Bergh, et al.Oncoimmunology|July 9, 2015
Extracellular vesicle-mediated transfer of functional RNA in the tumor microenvironmentKirsten Ridder, Alexandra Sevko, Janina Heide, et al.The EMBO Journal|August 18, 2011
Class IIb HDAC6 regulates endothelial cell migration and angiogenesis by deacetylation of cortactinDavid Kaluza, Jens Kroll, Sabine Gesierich, et al.Journal of the American College of Cardiology|July 21, 2012
Circulating apoptotic endothelial cells and apoptotic endothelial microparticles independently predict the presence of cardiac allograft vasculopathyNeha Singh, Eline Van Craeyveld, Marc Tjwa, et al.The Journal of Clinical Investigation|November 3, 2007
Myocardial hypertrophy in the absence of external stimuli is induced by angiogenesis in miceDaniela Tirziu, Emmanuel Chorianopoulos, Karen L Moodie, et al.Circulation Research|March 27, 2004
Cardiomyocyte-specific overexpression of nitric oxide synthase 3 improves left ventricular performance and reduces compensatory hypertrophy after myocardial infarctionStefan Janssens, Peter Pokreisz, Luc Schoonjans, et al.Pageof 6