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Marcel Doerflinger

Showing results (41-50 of 50) with videos related to

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Nature Communications|June 15, 2026
Efferocytosis of apoptotic bodies drives SARS-CoV-2 infection and macrophage inflammationThanh Kha Phan, Dylan Sheerin, Bo Shi, et al.
Immunity|August 1, 2020
Flexible Usage and Interconnectivity of Diverse Cell Death Pathways Protect against Intracellular InfectionMarcel Doerflinger, Yexuan Deng, Paul Whitney, et al.
The EMBO Journal|January 17, 2023
Caspase-8-driven apoptotic and pyroptotic crosstalk causes cell death and IL-1β release in X-linked inhibitor of apoptosis (XIAP) deficiencySebastian A Hughes, Meng Lin, Ashley Weir, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 24, 2021
Nanobody cocktails potently neutralize SARS-CoV-2 D614G N501Y variant and protect micePhillip Pymm, Amy Adair, Li-Jin Chan, et al.
Gastroenterology|August 29, 2022
Epigenetic Silencing of RIPK3 in Hepatocytes Prevents MLKL-mediated Necroptosis From Contributing to Liver PathologiesSimon P Preston, Michael D Stutz, Cody C Allison, et al.
The EMBO Journal|August 27, 2020
Mechanism and inhibition of the papain-like protease, PLpro, of SARS-CoV-2Theresa Klemm, Gregor Ebert, Dale J Calleja, et al.
Nature Communications|September 28, 2023
A common human MLKL polymorphism confers resistance to negative regulation by phosphorylationSarah E Garnish, Katherine R Martin, Maria Kauppi, et al.
Nature Communications|November 13, 2025
Non-apoptotic caspase-8 is critical for orchestrating exaggerated inflammation during severe SARS-CoV-2 infectionStefanie M Bader, Lena Scherer, Reet Bhandari, et al.
Immunity|February 9, 2022
Interferon-γ primes macrophages for pathogen ligand-induced killing via a caspase-8 and mitochondrial cell death pathwayDaniel S Simpson, Jiyi Pang, Ashley Weir, et al.
Nature Communications|April 3, 2025
A novel PLpro inhibitor improves outcomes in a pre-clinical model of long COVIDStefanie M Bader, Dale J Calleja, Shane M Devine, et al.
Pageof 5

Showing results (41-50 of 50) with videos related to

Sort By:
Pageof 5
You have reached the last page of results.This site can display upto 50 results.
Nature Communications|June 15, 2026
Efferocytosis of apoptotic bodies drives SARS-CoV-2 infection and macrophage inflammationThanh Kha Phan, Dylan Sheerin, Bo Shi, et al.
Immunity|August 1, 2020
Flexible Usage and Interconnectivity of Diverse Cell Death Pathways Protect against Intracellular InfectionMarcel Doerflinger, Yexuan Deng, Paul Whitney, et al.
The EMBO Journal|January 17, 2023
Caspase-8-driven apoptotic and pyroptotic crosstalk causes cell death and IL-1β release in X-linked inhibitor of apoptosis (XIAP) deficiencySebastian A Hughes, Meng Lin, Ashley Weir, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 24, 2021
Nanobody cocktails potently neutralize SARS-CoV-2 D614G N501Y variant and protect micePhillip Pymm, Amy Adair, Li-Jin Chan, et al.
Gastroenterology|August 29, 2022
Epigenetic Silencing of RIPK3 in Hepatocytes Prevents MLKL-mediated Necroptosis From Contributing to Liver PathologiesSimon P Preston, Michael D Stutz, Cody C Allison, et al.
The EMBO Journal|August 27, 2020
Mechanism and inhibition of the papain-like protease, PLpro, of SARS-CoV-2Theresa Klemm, Gregor Ebert, Dale J Calleja, et al.
Nature Communications|September 28, 2023
A common human MLKL polymorphism confers resistance to negative regulation by phosphorylationSarah E Garnish, Katherine R Martin, Maria Kauppi, et al.
Nature Communications|November 13, 2025
Non-apoptotic caspase-8 is critical for orchestrating exaggerated inflammation during severe SARS-CoV-2 infectionStefanie M Bader, Lena Scherer, Reet Bhandari, et al.
Immunity|February 9, 2022
Interferon-γ primes macrophages for pathogen ligand-induced killing via a caspase-8 and mitochondrial cell death pathwayDaniel S Simpson, Jiyi Pang, Ashley Weir, et al.
Nature Communications|April 3, 2025
A novel PLpro inhibitor improves outcomes in a pre-clinical model of long COVIDStefanie M Bader, Dale J Calleja, Shane M Devine, et al.
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