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The Journal of Investigative Dermatology
|
September 14, 2007
Five new CYLD mutations in skin appendage tumors and evidence that aspartic acid 681 in CYLD is essential for deubiquitinase activity
Stéphanie Almeida, Caroline Maillard, Peter Itin, et al.
The Journal of Investigative Dermatology
|
January 29, 2020
Palmoplantar Keratoderma with Leukokeratosis Anogenitalis Caused by KDSR Mutations
Marcel Huber, Elena Chiticariu, Daniel Bachmann, et al.
The Journal of Investigative Dermatology
|
November 12, 2010
The TRAF-interacting protein (TRIP) is a regulator of keratinocyte proliferation
Stéphanie Almeida, Stephan Ryser, Magdalena Obarzanek-Fojt, et al.
Revue Medicale Suisse
|
May 14, 2016
[The skin microbiota: a colossus steps into the spotlight]
Jeremy Di Domizio, Alessandra Pagnoni, Marcel Huber, et al.
The Journal of Investigative Dermatology
|
March 28, 2016
HOPX: The Unusual Homeodomain-Containing Protein
Anita Mariotto, Olesya Pavlova, Hyun-Sook Park, et al.
Biochemical and Biophysical Research Communications
|
September 26, 2006
Histone acetyltransferase HBO1 inhibits NF-kappaB activity by coactivator sequestration
Romuald Contzler, Alexander Regamey, Bertrand Favre, et al.
Journal of Cell Science
|
October 23, 2014
TRAIP is a regulator of the spindle assembly checkpoint
Christophe Chapard, Patrick Meraldi, Tobias Gleich, et al.
European Journal of Cell Biology
|
January 25, 2011
Homeodomain-only protein HOP is a novel modulator of late differentiation in keratinocytes
Magdalena Obarzanek-Fojt, Bertrand Favre, Magdalini Kypriotou, et al.
Journal of Cell Science
|
July 31, 2015
The caspase-3-p120-RasGAP module generates a NF-κB repressor in response to cellular stress
Hadi Khalil, Noureddine Loukili, Alexandre Regamey, et al.
The Journal of Experimental Medicine
|
December 17, 2003
The tumor suppressor CYLD interacts with TRIP and regulates negatively nuclear factor kappaB activation by tumor necrosis factor
Alexandre Regamey, Daniel Hohl, Jia Wei Liu, et al.
Page
of 4
Search research articles
Search
Showing results (11-20 of 39) with videos related to
Sort By:
Page
of 4
The Journal of Investigative Dermatology
|
September 14, 2007
Five new CYLD mutations in skin appendage tumors and evidence that aspartic acid 681 in CYLD is essential for deubiquitinase activity
Stéphanie Almeida, Caroline Maillard, Peter Itin, et al.
The Journal of Investigative Dermatology
|
January 29, 2020
Palmoplantar Keratoderma with Leukokeratosis Anogenitalis Caused by KDSR Mutations
Marcel Huber, Elena Chiticariu, Daniel Bachmann, et al.
The Journal of Investigative Dermatology
|
November 12, 2010
The TRAF-interacting protein (TRIP) is a regulator of keratinocyte proliferation
Stéphanie Almeida, Stephan Ryser, Magdalena Obarzanek-Fojt, et al.
Revue Medicale Suisse
|
May 14, 2016
[The skin microbiota: a colossus steps into the spotlight]
Jeremy Di Domizio, Alessandra Pagnoni, Marcel Huber, et al.
The Journal of Investigative Dermatology
|
March 28, 2016
HOPX: The Unusual Homeodomain-Containing Protein
Anita Mariotto, Olesya Pavlova, Hyun-Sook Park, et al.
Biochemical and Biophysical Research Communications
|
September 26, 2006
Histone acetyltransferase HBO1 inhibits NF-kappaB activity by coactivator sequestration
Romuald Contzler, Alexander Regamey, Bertrand Favre, et al.
Journal of Cell Science
|
October 23, 2014
TRAIP is a regulator of the spindle assembly checkpoint
Christophe Chapard, Patrick Meraldi, Tobias Gleich, et al.
European Journal of Cell Biology
|
January 25, 2011
Homeodomain-only protein HOP is a novel modulator of late differentiation in keratinocytes
Magdalena Obarzanek-Fojt, Bertrand Favre, Magdalini Kypriotou, et al.
Journal of Cell Science
|
July 31, 2015
The caspase-3-p120-RasGAP module generates a NF-κB repressor in response to cellular stress
Hadi Khalil, Noureddine Loukili, Alexandre Regamey, et al.
The Journal of Experimental Medicine
|
December 17, 2003
The tumor suppressor CYLD interacts with TRIP and regulates negatively nuclear factor kappaB activation by tumor necrosis factor
Alexandre Regamey, Daniel Hohl, Jia Wei Liu, et al.
Page
of 4