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Cellular and Molecular Life Sciences : CMLS|February 23, 2018
Aurora kinase B regulates axonal outgrowth and regeneration in the spinal motor neurons of developing zebrafishSerene S L Gwee, Rowan A W Radford, Sharron Chow, et al.Human Molecular Genetics|March 17, 2021
ALS/FTD-causing mutation in cyclin F causes the dysregulation of SFPQStephanie L Rayner, Flora Cheng, Alison L Hogan, et al.Neurobiology of Disease|March 1, 2022
TDP-43 is a ubiquitylation substrate of the SCFcyclin F complexStephanie L Rayner, Shu Yang, Natalie E Farrawell, et al.Journal of Neurochemistry|May 14, 2025
Proteomics Analysis of the TDP-43 Interactome in Cellular Models of ALS PathogenesisFlora Cheng, Tyler Chapman, Juliana Venturato, et al.The American Journal of Pathology|March 27, 2018
Selective Spatiotemporal Vulnerability of Central Nervous System Neurons to Pathologic TAR DNA-Binding Protein 43 in Aged Transgenic MiceAnnika van Hummel, Gabriella Chan, Julia van der Hoven, et al.Molecular Neurobiology|January 8, 2021
In vivo Validation of Bimolecular Fluorescence Complementation (BiFC) to Investigate Aggregate Formation in Amyotrophic Lateral Sclerosis (ALS)Emily K Don, Alina Maschirow, Rowan A W Radford, et al.Journal of Neuroinflammation|May 2, 2026
ALS-FTD-linked CCNFS621G drives increased hippocampal astrocyte ramification and mitochondrial dysfunction and impairs motor neuron excitabilityLiam Robinson, Dzung Do-Ha, Flora Cheng, et al.The European Journal of Neuroscience|August 14, 2021
Riluzole does not ameliorate disease caused by cytoplasmic TDP-43 in a mouse model of amyotrophic lateral sclerosisAmanda L Wright, Paul A Della Gatta, Sheng Le, et al.Neuropathology and Applied Neurobiology|July 21, 2021
Splicing factor proline and glutamine rich intron retention, reduced expression and aggregate formation are pathological features of amyotrophic lateral sclerosisAlison L Hogan, Natalie Grima, Jennifer A Fifita, et al.Molecular Neurobiology|May 27, 2023
The E3 Ubiquitin Ligase SCF Cyclin F Promotes Sequestosome-1/p62 Insolubility and Foci Formation and is Dysregulated in ALS and FTD PathogenesisJennilee M Davidson, Sharlynn S L Wu, Stephanie L Rayner, et al.Pageof 18