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Marco Pontoglio

Showing results (41-50 of 57) with videos related to

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Science Translational Medicine|June 25, 2025
Podocyte-specific YAP activation triggers crescentic glomerulonephritis through cross-talk with parietal epithelial cellsLisa Turinsky, Sonja Rehrl, Clément Nguyen, et al.
Hepatology (Baltimore, Md.)|June 19, 2012
HNF1B deficiency causes ciliary defects in human cholangiocytesPhilip Roelandt, Aline Antoniou, Louis Libbrecht, et al.
Nature Communications|April 16, 2021
Single cell regulatory landscape of the mouse kidney highlights cellular differentiation programs and disease targetsZhen Miao, Michael S Balzer, Ziyuan Ma, et al.
Journal of the American Society of Nephrology : JASN|July 26, 2017
Hepatocyte Nuclear Factor-1<i>β</i> Controls Mitochondrial Respiration in Renal Tubular CellsAudrey Casemayou, Audren Fournel, Alessia Bagattin, et al.
JCI Insight|May 8, 2020
Signaling pathways predisposing to chronic kidney disease progressionMohamad Zaidan, Martine Burtin, Jitao David Zhang, et al.
Journal of the American Society of Nephrology : JASN|May 8, 2016
Stat3 Controls Tubulointerstitial Communication during CKDFrank Bienaimé, Mordi Muorah, Lucie Yammine, et al.
Plos Genetics|December 15, 2017
MITF - A controls branching morphogenesis and nephron endowmentAurélie Phelep, Denise Laouari, Kapil Bharti, et al.
Kidney International|April 17, 2016
Tubular proteinuria in patients with HNF1α mutations: HNF1α drives endocytosis in the proximal tubuleSara Terryn, Karo Tanaka, Jean-Philippe Lengelé, et al.
Nature Medicine|September 24, 2013
AKT2 is essential to maintain podocyte viability and function during chronic kidney diseaseGuillaume Canaud, Frank Bienaimé, Amandine Viau, et al.
Hepatology (Baltimore, Md.)|March 11, 2011
A classification of ductal plate malformations based on distinct pathogenic mechanisms of biliary dysmorphogenesisPeggy Raynaud, Joshua Tate, Céline Callens, et al.
Pageof 6

Showing results (41-50 of 57) with videos related to

Sort By:
Pageof 6
Science Translational Medicine|June 25, 2025
Podocyte-specific YAP activation triggers crescentic glomerulonephritis through cross-talk with parietal epithelial cellsLisa Turinsky, Sonja Rehrl, Clément Nguyen, et al.
Hepatology (Baltimore, Md.)|June 19, 2012
HNF1B deficiency causes ciliary defects in human cholangiocytesPhilip Roelandt, Aline Antoniou, Louis Libbrecht, et al.
Nature Communications|April 16, 2021
Single cell regulatory landscape of the mouse kidney highlights cellular differentiation programs and disease targetsZhen Miao, Michael S Balzer, Ziyuan Ma, et al.
Journal of the American Society of Nephrology : JASN|July 26, 2017
Hepatocyte Nuclear Factor-1<i>β</i> Controls Mitochondrial Respiration in Renal Tubular CellsAudrey Casemayou, Audren Fournel, Alessia Bagattin, et al.
JCI Insight|May 8, 2020
Signaling pathways predisposing to chronic kidney disease progressionMohamad Zaidan, Martine Burtin, Jitao David Zhang, et al.
Journal of the American Society of Nephrology : JASN|May 8, 2016
Stat3 Controls Tubulointerstitial Communication during CKDFrank Bienaimé, Mordi Muorah, Lucie Yammine, et al.
Plos Genetics|December 15, 2017
MITF - A controls branching morphogenesis and nephron endowmentAurélie Phelep, Denise Laouari, Kapil Bharti, et al.
Kidney International|April 17, 2016
Tubular proteinuria in patients with HNF1α mutations: HNF1α drives endocytosis in the proximal tubuleSara Terryn, Karo Tanaka, Jean-Philippe Lengelé, et al.
Nature Medicine|September 24, 2013
AKT2 is essential to maintain podocyte viability and function during chronic kidney diseaseGuillaume Canaud, Frank Bienaimé, Amandine Viau, et al.
Hepatology (Baltimore, Md.)|March 11, 2011
A classification of ductal plate malformations based on distinct pathogenic mechanisms of biliary dysmorphogenesisPeggy Raynaud, Joshua Tate, Céline Callens, et al.
Pageof 6