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Plos One|November 9, 2013
Transcriptional reprogramming of CD11b+Esam(hi) dendritic cell identity and function by loss of Runx3Joseph Dicken, Alexander Mildner, Dena Leshkowitz, et al.Frontiers in Immunology|May 31, 2021
GPCRomics of Homeostatic and Disease-Associated Human MicrogliaCheng-Chih Hsiao, Roman Sankowski, Marco Prinz, et al.Stem Cell Reports|December 29, 2023
C/EBPβ-induced lymphoid-to-myeloid transdifferentiation emulates granulocyte-monocyte progenitor biologyLinh Thuy Nguyen, Karin Zimmermann, Elisabeth Kowenz-Leutz, et al.European Journal of Immunology|May 13, 2022
Paradoxical immunodeficiencies-When failures of innate immunity cause immunopathologyJulia Kolter, Philipp Henneke, Olaf Groß, et al.Cochlear Implants International|February 21, 2012
Necrotizing meningoencephalitis mimicking cerebellopontine angle tumor as late complication following cochlear implantationSusan Arndt, Christian Schild, Soroush Doostkam, et al.The Journal of Antimicrobial Chemotherapy|November 3, 2006
Minocycline delays but does not attenuate the course of experimental autoimmune encephalomyelitis in Streptococcus pneumoniae-infected miceIsabel Herrmann, Markus Kellert, Annette Spreer, et al.Experimental Neurology|January 6, 2009
Type I interferon receptor signalling is induced during demyelination while its function for myelin damage and repair is redundantHauke Schmidt, Jenni Raasch, Doron Merkler, et al.Nature Communications|April 25, 2017
Neuronal IFN-beta-induced PI3K/Akt-FoxA1 signalling is essential for generation of FoxA1+Treg cellsYawei Liu, Andrea Marin, Patrick Ejlerskov, et al.Brain : a Journal of Neurology|April 29, 2010
Induction of inhibitory central nervous system-derived and stimulatory blood-derived dendritic cells suggests a dual role for granulocyte-macrophage colony-stimulating factor in central nervous system inflammationLysann Hesske, Christine Vincenzetti, Mathias Heikenwalder, et al.Brain Structure & Function|February 2, 2019
Microglia contribute to the glia limitans around arteries, capillaries and veins under physiological conditions, in a model of neuroinflammation and in human brain tissueEmely Joost, Marta J C Jordão, Bianca Mages, et al.Pageof 45