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Cell Cycle (Georgetown, Tex.)|October 12, 2010
The dark side of fly TNF: an ancient developmental proof reading mechanism turned into tumor promoterMarcos VidalCell Adhesion & Migration|August 13, 2011
Epithelial delamination and migration: lessons from DrosophilaFederica Parisi, Marcos VidalCurrent Opinion in Genetics & Development|December 20, 2005
Drosophila models for cancer researchMarcos Vidal, Ross L CaganJournal of Genetics and Genomics = Yi Chuan Xue Bao|November 1, 2011
Tumor invasion and metastasis in Drosophila: a bold past, a bright futureRhoda K A Stefanatos, Marcos VidalCell Cycle (Georgetown, Tex.)|August 7, 2009
APC as a master regulator of intestinal homeostasis and transformation: from flies to vertebratesJulia Cordero, Marcos Vidal, Owen SansomPlos One|December 19, 2013
p120 catenin is required for the stress response in DrosophilaRhoda K Stefanatos, Christin Bauer, Marcos VidalCancer Research|May 4, 2005
ZD6474 suppresses oncogenic RET isoforms in a Drosophila model for type 2 multiple endocrine neoplasia syndromes and papillary thyroid carcinomaMarcos Vidal, Samuel Wells, Anderson Ryan, et al.Developmental Cell|January 10, 2006
Csk-deficient boundary cells are eliminated from normal Drosophila epithelia by exclusion, migration, and apoptosisMarcos Vidal, David E Larson, Ross L CaganMolecular and Cellular Biology|March 23, 2002
Stat1-vitamin D receptor interactions antagonize 1,25-dihydroxyvitamin D transcriptional activity and enhance stat1-mediated transcriptionMarcos Vidal, Chilakamarti V Ramana, Adriana S DussoCancer Research|November 3, 2007
Differing Src signaling levels have distinct outcomes in DrosophilaMarcos Vidal, Stephen Warner, Renee Read, et al.Pageof 10