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Mari Masuda

Showing results (41-50 of 44) with videos related to

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Cancer Research|July 2, 2020
Extracellular Vesicles from Cancer-Associated Fibroblasts Containing Annexin A6 Induces FAK-YAP Activation by Stabilizing β1 Integrin, Enhancing Drug ResistanceTomoyuki Uchihara, Keisuke Miyake, Atsuko Yonemura, et al.
Annals of Surgery Open : Perspectives of Surgical History, Education, and Clinical Approaches|March 26, 2025
Impact of Sensitive Circulating Tumor DNA Monitoring on CT Scan Intervals During Postoperative Colorectal Cancer SurveillanceTomoko Sasaki, Takeshi Iwaya, Mizunori Yaegashi, et al.
Cancer Research|December 21, 2022
Stromal Reprogramming through Dual PDGFRα/β Blockade Boosts the Efficacy of Anti-PD-1 Immunotherapy in Fibrotic TumorsTakahiko Akiyama, Tadahito Yasuda, Tomoyuki Uchihara, et al.
Molecular Cancer|June 11, 2024
Mechanism of ERBB2 gene overexpression by the formation of super-enhancer with genomic structural abnormalities in lung adenocarcinoma without clinically actionable genetic alterationsSyuzo Kaneko, Ken Takasawa, Ken Asada, et al.
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Showing results (41-50 of 44) with videos related to

Sort By:
Pageof 5
You have reached the last page of results.This site can display upto 44 results.
Cancer Research|July 2, 2020
Extracellular Vesicles from Cancer-Associated Fibroblasts Containing Annexin A6 Induces FAK-YAP Activation by Stabilizing β1 Integrin, Enhancing Drug ResistanceTomoyuki Uchihara, Keisuke Miyake, Atsuko Yonemura, et al.
Annals of Surgery Open : Perspectives of Surgical History, Education, and Clinical Approaches|March 26, 2025
Impact of Sensitive Circulating Tumor DNA Monitoring on CT Scan Intervals During Postoperative Colorectal Cancer SurveillanceTomoko Sasaki, Takeshi Iwaya, Mizunori Yaegashi, et al.
Cancer Research|December 21, 2022
Stromal Reprogramming through Dual PDGFRα/β Blockade Boosts the Efficacy of Anti-PD-1 Immunotherapy in Fibrotic TumorsTakahiko Akiyama, Tadahito Yasuda, Tomoyuki Uchihara, et al.
Molecular Cancer|June 11, 2024
Mechanism of ERBB2 gene overexpression by the formation of super-enhancer with genomic structural abnormalities in lung adenocarcinoma without clinically actionable genetic alterationsSyuzo Kaneko, Ken Takasawa, Ken Asada, et al.
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